Specific Inhibition of Complement Activation Significantly Ameliorates Autoimmune Blistering Disease in Mice.

about

Specific Inhibition of Complement Activation Significantly Ameliorates Autoimmune Blistering Disease in Mice.

Item

http://www.wikidata.org/entity/Q52332743

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@en

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@nl

type

Item

label

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@en

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@nl

prefLabel

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@en

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@nl

P1476

Specific Inhibition of Complem ...... ne Blistering Disease in Mice.

@en

P2093

Detlef Zillikens

http://www.w3.org/2001/XMLSchema#string

Falk Nimmerjahn

http://www.w3.org/2001/XMLSchema#string

Joshua M Thurman

http://www.w3.org/2001/XMLSchema#string

Jörg Köhl

http://www.w3.org/2001/XMLSchema#string

Misa Hirose

http://www.w3.org/2001/XMLSchema#string

Sidonia Mihai

http://www.w3.org/2001/XMLSchema#string

V Michael Holers

http://www.w3.org/2001/XMLSchema#string

Yi Wang

http://www.w3.org/2001/XMLSchema#string

P2860

C5L2: a controversial receptor of complement anaphylatoxin, C5a

C5a mutants are potent antagonists of the C5a receptor (CD88) and of C5L2: position 69 is the locus that determines agonism or antagonism

Complement: a key system for immune surveillance and homeostasis

Induction of dermal-epidermal separation in mice by passive transfer of antibodies specific to type VII collagen.

Mechanisms of Autoantibody-Induced Pathology.

Anti-C5 monoclonal antibody therapy prevents collagen-induced arthritis and ameliorates established disease.

Role of C3a receptors, C5a receptors, and complement protein C6 deficiency in collagen antibody-induced arthritis in mice.

The role of complement in experimental bullous pemphigoid.

The complement inhibitor eculizumab in paroxysmal nocturnal hemoglobinuria.

Neutrophil-endothelial cell interactions. Modulation of neutrophil adhesiveness induced by complement fragments C5a and C5a des arg and formyl-methionyl-leucyl-phenylalanine in vitro.

P304

535

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3389/FIMMU.2018.00535

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Ralf J. Ludwig

Paul Morgan

P577

2018-03-16T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29616034

http://www.w3.org/2001/XMLSchema#string

P932

5865061

http://www.w3.org/2001/XMLSchema#string