about
Hepatitis C virus (HCV) induces formation of stress granules whose proteins regulate HCV RNA replication and virus assembly and egress.Genetic analysis of influenza virus NS1 gene: a temperature-sensitive mutant shows defective formation of virus particles.Plasmacytoid dendritic cells sense hepatitis C virus-infected cells, produce interferon, and inhibit infection.Hepatitis C virus blocks interferon effector function by inducing protein kinase R phosphorylation.Short-range exosomal transfer of viral RNA from infected cells to plasmacytoid dendritic cells triggers innate immunity.Mutation analysis of a recombinant NS replicon shows that influenza virus NS1 protein blocks the splicing and nucleo-cytoplasmic transport of its own viral mRNA.In vivo reduction of hepatitis B virus antigenemia and viremia by antisense oligonucleotides.Emergent properties of the interferon-signalling network may underlie the success of hepatitis C treatment.Hepatitis B Virus and DNA Damage Response: Interactions and Consequences for the Infection.HBsAg-redirected T cells exhibit antiviral activity in HBV-infected human liver chimeric mice.
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description
researcher
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wetenschapper
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հետազոտող
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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Urtzi Garaigorta
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10640610900
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P496
0000-0002-0683-5725