Disturbed connexin43 gap junction distribution correlates with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardia.
about
A novel desmocollin-2 mutation reveals insights into the molecular link between desmosomes and gap junctionsConnexin43 interacts with Caveolin-3 in the heartReduced expression of Cx43 attenuates ventricular remodeling after myocardial infarction via impaired TGF-beta signalingComputational model of erratic arrhythmias in a cardiac cell network: the role of gap junctionsCharacterization of the Structure and Intermolecular Interactions between the Connexin40 and Connexin43 Carboxyl-terminal and Cytoplasmic Loop DomainsNew functions for alpha-catenins in health and disease: from cancer to heart regenerationConduction slowing and sudden arrhythmic death in mice with cardiac-restricted inactivation of connexin43Mechanoelectrical remodeling and arrhythmias during progression of hypertrophy.Structural and molecular mechanisms of gap junction remodeling in epicardial border zone myocytes following myocardial infarctionpH-dependent dimerization of the carboxyl terminal domain of Cx43.Making better scar: Emerging approaches for modifying mechanical and electrical properties following infarction and ablationGenesis of ectopic waves: role of coupling, automaticity, and heterogeneityMicrotubule plus-end-tracking proteins target gap junctions directly from the cell interior to adherens junctionsLimited forward trafficking of connexin 43 reduces cell-cell coupling in stressed human and mouse myocardium.Improving cardiac conduction with a skeletal muscle sodium channel by gene and cell therapyActin cytoskeleton rest stops regulate anterograde traffic of connexin 43 vesicles to the plasma membrane.Up-regulation of heme oxygenase-1 after infarct initiation reduces mortality, infarct size and left ventricular remodeling: experimental evidence and proof of concept.Altered pattern of connexin40 distribution in persistent atrial fibrillation in the goat.Inherited long QT syndromes: a paradigm for understanding arrhythmogenesis.Changes in connexin 43, metalloproteinase and tissue inhibitor of metalloproteinase during tachycardia-induced cardiomyopathy in dogs.Short-term pacing in the mouse alters cardiac expression of connexin43.Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis.Phosphorylation of connexin43 on serine 306 regulates electrical coupling.Multiple monophasic shocks improve electrotherapy of ventricular tachycardia in a rabbit model of chronic infarction.The role of transmural ventricular heterogeneities in cardiac vulnerability to electric shocks.Mechanisms for initiation of reentry in acute regional ischemia phase 1B.Gene therapies for arrhythmias in heart failure.The cardiac muscle cell.Electrophysiological remodeling in heart failureGap junction remodeling in infarction: does it play a role in arrhythmogenesis?Remodeling of cardiac fibroblasts following myocardial infarction results in increased gap junction intercellular communicationThe impact of recent ion channel science on the development and use of antiarrhythmic drugs.The Renin-Angiotensin system mediates the effects of stretch on conduction velocity, connexin43 expression, and redistribution in intact ventricle.Immunocytochemical analysis of connexin expression in the healthy and diseased cardiovascular system.Longitudinal arrhythmogenic remodelling in a mouse model of longstanding pressure overload.Modulation of cardiac gap junction expression and arrhythmic susceptibility.Constitutively active calcineurin induces cardiac endoplasmic reticulum stress and protects against apoptosis that is mediated by alpha-crystallin-B.Ischemia-related subcellular redistribution of sodium channels enhances the proarrhythmic effect of class I antiarrhythmic drugs: a simulation study.Ionic mechanisms of electrophysiological heterogeneity and conduction block in the infarct border zone.Spatiotemporal induction of matrix metalloproteinase-9 transcription after discrete myocardial injury.
P2860
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P2860
Disturbed connexin43 gap junction distribution correlates with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardia.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh-hant
name
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@en
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@nl
type
label
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@en
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@nl
prefLabel
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@en
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@nl
P2093
P356
P1433
P1476
Disturbed connexin43 gap junct ...... cause ventricular tachycardia.
@en
P2093
P304
P356
10.1161/01.CIR.95.4.988
P407
P577
1997-02-01T00:00:00Z