CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
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Alzheimer's disease as homeostatic responses to age-related myelin breakdownA putative functional role for oligodendrocytes in mood regulationProteolipid protein is required for transport of sirtuin 2 into CNS myelinDisruption of axo-glial junctions causes cytoskeletal disorganization and degeneration of Purkinje neuron axonsMyelin-specific proteins: a structurally diverse group of membrane-interacting molecules.The comprehensive native interactome of a fully functional tagged prion proteinNo relationship between 2',3'-cyclic nucleotide 3'-phosphodiesterase and schizophrenia in the Chinese Han population: an expression study and meta-analysisThe encephalopathy of prematurity--brain injury and impaired brain development inextricably intertwined.Expression, purification, and initial characterization of different domains of recombinant mouse 2',3'-cyclic nucleotide 3'-phosphodiesterase, an enigmatic enzyme from the myelin sheath.Spatiotemporal ablation of myelinating glia-specific neurofascin (Nfasc NF155) in mice reveals gradual loss of paranodal axoglial junctions and concomitant disorganization of axonal domains.Myelin loss does not lead to axonal degeneration in a long-lived model of chronic demyelinationAxon-glial interaction in the CNS: what we have learned from mouse models of Pelizaeus-Merzbacher disease.Proteolipid protein cannot replace P0 protein as the major structural protein of peripheral nervous system myelin.Spatial mapping of protein abundances in the mouse brain by voxelation integrated with high-throughput liquid chromatography-mass spectrometryResilient emotionality and molecular compensation in mice lacking the oligodendrocyte-specific gene Cnp1Neonatal hyperoxia exposure disrupts axon-oligodendrocyte integrity in the subcortical white matter.Brain injury in premature infants: a complex amalgam of destructive and developmental disturbances.Myelin proteomics: molecular anatomy of an insulating sheath.Proteome of synaptosome-associated proteins in spinal cord dorsal horn after peripheral nerve injury.Phylogeny of proteolipid proteins: divergence, constraints, and the evolution of novel functions in myelination and neuroprotection.Early structural and functional defects in synapses and myelinated axons in stratum lacunosum moleculare in two preclinical models for tauopathy.The multiple roles of myelin protein genes during the development of the oligodendrocyte.Proteomic Analysis of Mitochondria-Enriched Fraction Isolated from the Frontal Cortex and Hippocampus of Apolipoprotein E Knockout Mice Treated with Alda-1, an Activator of Mitochondrial Aldehyde Dehydrogenase (ALDH2)Myelin restoration: progress and prospects for human cell replacement therapies.Enriched environment and white matter in aging brain.Oligodendroglia: metabolic supporters of axons.A unified cell biological perspective on axon-myelin injury.Oligodendroglia: metabolic supporters of neurons.Cerebrospinal fluid and blood biomarkers of neuroaxonal damage in multiple sclerosis.zRICH, a protein induced during optic nerve regeneration in zebrafish, promotes neuritogenesis and interacts with tubulin.JCV agnoprotein-induced reduction in CXCL5/LIX secretion by oligodendrocytes is associated with activation of apoptotic signaling in neurons.Novel molecular insights into the critical role of sulfatide in myelin maintenance/function.Simvastatin interferes with process outgrowth and branching of oligodendrocytes.Ultrastructural alterations of myelinated fibers and oligodendrocytes in the prefrontal cortex in schizophrenia: a postmortem morphometric study.Antagonistic Functions of MBP and CNP Establish Cytosolic Channels in CNS Myelin.Process outgrowth in oligodendrocytes is mediated by CNP, a novel microtubule assembly myelin protein.The quest for self-identity: not all cancer stem cells are the same.Polyomavirus JC infection inhibits differentiation of oligodendrocyte progenitor cells.Neuroinflammation in white matter tracts of Cnp1 mutant mice amplified by a minor brain injury.Regulation and dysregulation of axon infrastructure by myelinating glia.
P2860
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P2860
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@en
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@nl
type
label
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@en
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@nl
prefLabel
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@en
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@nl
P2093
P50
P356
P1433
P1476
CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.
@en
P2093
Jane Tayler
Rashmi Bansal
Yoshimi Kaga
P356
10.1002/GLIA.20165
P577
2005-04-01T00:00:00Z