Enteropathogenic Escherichia coli Tir translocation and pedestal formation requires membrane cholesterol in the absence of bundle-forming pili.
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Type Three Secretion System in Attaching and Effacing PathogensCytosolic extract induces Tir translocation and pedestals in EPEC-infected red blood cellsProtein export according to schedule: architecture, assembly, and regulation of type III secretion systems from plant- and animal-pathogenic bacteria.Bundle-forming pilus retraction enhances enteropathogenic Escherichia coli infectivityCytoskeleton-modulating effectors of enteropathogenic and enterohemorrhagic Escherichia coli: role of EspL2 in adherence and an alternative pathway for modulating cytoskeleton through Annexin A2 function.Gene Ontology annotation highlights shared and divergent pathogenic strategies of type III effector proteins deployed by the plant pathogen Pseudomonas syringae pv tomato DC3000 and animal pathogenic Escherichia coli strainsEnterohemorrhagic E. coli requires N-WASP for efficient type III translocation but not for EspFU-mediated actin pedestal formation.E. coli secreted protein F promotes EPEC invasion of intestinal epithelial cells via an SNX9-dependent mechanism.Helicobacter pylori exploits cholesterol-rich microdomains for induction of NF-kappaB-dependent responses and peptidoglycan delivery in epithelial cellsThe N-terminal amphipathic region of the Escherichia coli type III secretion system protein EspD is required for membrane insertion and functionDisruption of CFTR-dependent lipid rafts reduces bacterial levels and corneal disease in a murine model of Pseudomonas aeruginosa keratitisEnteropathogenic Escherichia coli subverts phosphatidylinositol 4,5-bisphosphate and phosphatidylinositol 3,4,5-trisphosphate upon epithelial cell infectionPathogenic bacteria induce colonic PepT1 expression: an implication in host defense response.The Bordetella type III secretion system effector BteA contains a conserved N-terminal motif that guides bacterial virulence factors to lipid rafts.Cytoskeleton-modulating effectors of enteropathogenic and enterohaemorrhagic Escherichia coli: Tir, EspFU and actin pedestal assembly.Membrane targeting and pore formation by the type III secretion system translocon.Hijacking and Use of Host Lipids by Intracellular Pathogens.Protein kinase C mediates enterohemorrhagic Escherichia coli O157:H7-induced attaching and effacing lesions.Type III Secreted Virulence Factors Manipulating Signaling to Actin Dynamics.Escherichia coli-induced epithelial hyporesponsiveness to secretagogues is associated with altered CFTR localization.Detergent-resistant microdomains mediate activation of host cell signaling in response to attaching-effacing bacteria.Amino acid residues within enterohemorrhagic Escherichia coli O157:H7 Tir involved in phosphorylation, alpha-actinin recruitment, and Nck-independent pedestal formation.EspF Interacts with nucleation-promoting factors to recruit junctional proteins into pedestals for pedestal maturation and disruption of paracellular permeability.Pore-forming Activity of the Escherichia coli Type III Secretion System Protein EspD.
P2860
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P2860
Enteropathogenic Escherichia coli Tir translocation and pedestal formation requires membrane cholesterol in the absence of bundle-forming pili.
description
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name
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@en
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@nl
type
label
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@en
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@nl
prefLabel
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@en
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@nl
P2093
P2860
P1476
Enteropathogenic Escherichia c ...... bsence of bundle-forming pili.
@en
P2093
Barbara Waddell
Emma Allen-Vercoe
Julie Deans
Rebekah DeVinney
Scott Livingstone
P2860
P304
P356
10.1111/J.1462-5822.2005.00654.X
P577
2006-04-01T00:00:00Z