Effects of NVP-BEZ235, a dual phosphatidylinositol 3-kinase/mammalian target of rapamycin inhibitor, on HTLV-1-infected T-cell lines.

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Effects of NVP-BEZ235, a dual phosphatidylinositol 3-kinase/mammalian target of rapamycin inhibitor, on HTLV-1-infected T-cell lines.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

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2018年學術文章

@yue

2018年學術文章

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name

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

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Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@nl

type

Item

label

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@en

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@nl

prefLabel

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@en

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@nl

P1476

Effects of NVP-BEZ235, a dual ...... HTLV-1-infected T-cell lines.

@en

P2093

Chie Ishikawa

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Masachika Senba

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Naoki Mori

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P2860

The retinoblastoma protein and cell cycle control

mTOR inhibition induces upstream receptor tyrosine kinase signaling and activates Akt

Targeting the phosphoinositide 3-kinase pathway in cancer

Identification and characterization of NVP-BEZ235, a new orally available dual phosphatidylinositol 3-kinase/mammalian target of rapamycin inhibitor with potent in vivo antitumor activity

Dual inhibition of PI3K and mTOR inhibits autocrine and paracrine proliferative loops in PI3K/Akt/mTOR-addicted lymphomas.

Two hits are better than one: targeting both phosphatidylinositol 3-kinase and mammalian target of rapamycin as a therapeutic strategy for acute leukemia treatment.

Novel phosphatidylinositol 3-kinase inhibitor NVP-BKM120 induces apoptosis in myeloma cells and shows synergistic anti-myeloma activity with dexamethasone.

The dual PI3K/mTOR inhibitor, NVP-BEZ235, is efficacious against follicular lymphoma

International peripheral T-cell and natural killer/T-cell lymphoma study: pathology findings and clinical outcomes.

Monoclonal integration of human T-cell leukemia provirus in all primary tumors of adult T-cell leukemia suggests causative role of human T-cell leukemia virus in the disease

P304

5311-5317

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scholarly article

P356

10.3892/OL.2018.7979

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P433

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P478

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2018-02-07T00:00:00Z

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29552172

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5840529

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