Inhibition of TRPA1 Attenuates Doxorubicin-Induced Acute Cardiotoxicity by Suppressing Oxidative Stress, the Inflammatory Response, and Endoplasmic Reticulum Stress.

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Inhibition of TRPA1 Attenuates Doxorubicin-Induced Acute Cardiotoxicity by Suppressing Oxidative Stress, the Inflammatory Response, and Endoplasmic Reticulum Stress.

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http://www.wikidata.org/entity/Q52717628

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2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@en

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@nl

type

Item

label

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@en

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@nl

prefLabel

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@en

Inhibition of TRPA1 Attenuates ...... Endoplasmic Reticulum Stress.

@nl

P1476

Inhibition of TRPA1 Attenuates ...... d Endoplasmic Reticulum Stress

@en

P2093

Di Ye

http://www.w3.org/2001/XMLSchema#string

Huimin Jiang

http://www.w3.org/2001/XMLSchema#string

Jianfang Liu

http://www.w3.org/2001/XMLSchema#string

Jing Ye

http://www.w3.org/2001/XMLSchema#string

Menglong Wang

http://www.w3.org/2001/XMLSchema#string

Yao Xu

http://www.w3.org/2001/XMLSchema#string

Zhen Wang

http://www.w3.org/2001/XMLSchema#string

P2860

TRPA1 mediates the inflammatory actions of environmental irritants and proalgesic agents

ROS function in redox signaling and oxidative stress

Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress

Chemical Endoplasmic Reticulum Chaperone Alleviates Doxorubicin-Induced Cardiac Dysfunction.

Free radicals and antioxidants: updating a personal view.

Nox2 NADPH oxidase promotes pathologic cardiac remodeling associated with Doxorubicin chemotherapy.

NLRP3 Deficiency Reduces Macrophage Interleukin-10 Production and Enhances the Susceptibility to Doxorubicin-induced Cardiotoxicity.

Early detection and prediction of cardiotoxicity in chemotherapy-treated patients

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Over-expression of a modified bifunctional apoptosis regulator protects against cardiac injury and doxorubicin-induced cardiotoxicity in transgenic mice

P304

5179468

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P31

scholarly article

P356

10.1155/2018/5179468

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P478

2018

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P50

Jun Wan

P577

2018-02-28T00:00:00Z

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P698

29682158

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P921

endoplasmic reticulum

P932

5850896

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