EBV persistence without its EBNA3A and 3C oncogenes in vivo.

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An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model

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Q57025486-B4BFAFE1-C26E-452E-B64A-6CF0D6C4AAE4

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EBV persistence without its EBNA3A and 3C oncogenes in vivo.

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http://www.wikidata.org/entity/Q52803943

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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name

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@en

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@nl

type

Item

label

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@en

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@nl

prefLabel

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@en

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@nl

P1476

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

@en

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Andrea Zbinden

http://www.w3.org/2001/XMLSchema#string

Anita Murer

http://www.w3.org/2001/XMLSchema#string

Donal McHugh

http://www.w3.org/2001/XMLSchema#string

Gerald Niedobitek

http://www.w3.org/2001/XMLSchema#string

Jens Kalchschmidt

http://www.w3.org/2001/XMLSchema#string

Mario Barros

http://www.w3.org/2001/XMLSchema#string

Martin Allday

http://www.w3.org/2001/XMLSchema#string

P2860

Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2.

Priming of protective T cell responses against virus-induced tumors in mice with human immune system components

MORPHOLOGICAL AND BIOLOGICAL STUDIES ON A VIRUS IN CULTURED LYMPHOBLASTS FROM BURKITT'S LYMPHOMA

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

Adoptive transfer of EBV specific CD8+ T cell clones can transiently control EBV infection in humanized mice

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

IL-21 imposes a type II EBV gene expression on type III and type I B cells by the repression of C- and activation of LMP-1-promoter.

Epstein-barr virus nuclear antigen 3C activates the latent membrane protein 1 promoter in the presence of Epstein-Barr virus nuclear antigen 2 through sequences encompassing an spi-1/Spi-B binding site.

VIRUS PARTICLES IN CULTURED LYMPHOBLASTS FROM BURKITT'S LYMPHOMA.

The effect of Epstein-Barr virus Latent Membrane Protein 2 expression on the kinetics of early B cell infection

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e1007039

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scholarly article

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10.1371/JOURNAL.PPAT.1007039

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2018-04-30T00:00:00Z

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29709016

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P932

5945050

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EBV persistence without its EBNA3A and 3C oncogenes in vivo.

about

P2860

P2860

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P932