about
Evidence for recombination of live, attenuated immunodeficiency virus vaccine with challenge virus to a more virulent strain.Host factors determine differential disease progression after infection with nef-deleted simian immunodeficiency virusThe side population of ovarian cancer cells defines a heterogeneous compartment exhibiting stem cell characteristicsThe pathogenesis of HIV-induced dementia.Parkinsonism in HIV dementia.Psychiatric complications in human immunodeficiency virus infection.Increased APOBEC3G and APOBEC3F expression is associated with low viral load and prolonged survival in simian immunodeficiency virus infected rhesus monkeys.HIV-infection of the central nervous system: the tightrope walk of innate immunity.Drug Transporter-Mediated Protection of Cancer Stem Cells From Ionophore Antibiotics.On-demand erythrocyte disposal and iron recycling requires transient macrophages in the liverIncreases in CSF dopamine in HIV patients are due to the dopamine transporter 10/10-repeat allele which is more frequent in HIV-infected individuals.Ovarian cancer stem cells.Modulation of Immune Cell Functions by the E3 Ligase Cbl-b.Treg(s) in Cancer: Friends or Foe?The influence of stromal cells and tumor-microenvironment-derived cytokines and chemokines on CD3+CD8+ tumor infiltrating lymphocyte subpopulations.Opiate receptor blockade on human granulosa cells inhibits VEGF release.Identification of molecular sub-networks associated with cell survival in a chronically SIVmac-infected human CD4+ T cell line.Protective role of the virus-specific immune response for development of severe neurologic signs in simian immunodeficiency virus-infected macaques.Construction, replication, and immunogenic properties of a simian immunodeficiency virus expressing interleukin-2.Caspase inhibitors as a supplement in immune activation therapies to achieve eradication of HIV in its latent reservoirs.Caspase inhibition activates HIV in latently infected cells. Role of tumor necrosis factor receptor 1 and CD95.Dopamine activates HIV in chronically infected T lymphoblasts.U-373 MG glioblastoma and IMR-32 neuroblastoma cell lines express the dopamine and vesicular monoamine transporters.Stable multi-infection of splenocytes during SIV infection--the basis for continuous recombination.CD4 deficits and disease course acceleration can be driven by a collapse of the CD8 response in rhesus macaques infected with simian immunodeficiency virus.Early impairment in dopaminergic neurotransmission in brains of SIV-infected rhesus monkeys due to microglia activation.Azacytidine impairs NK cell activity in AML and MDS patients undergoing MRD-based pre-emptive treatment after allogeneic stem cell transplantationMicrobial translocation in simian immunodeficiency virus (SIV)-infected rhesus monkeys (Macaca mulatta).Relationship between viral load in blood, cerebrospinal fluid, brain tissue and isolated microglia with neurological disease in macaques infected with different strains of SIV.Intrathecal viral replication and cerebral deficits in different stages of human immunodeficiency virus disease.Impact of simian immunodeficiency virus (SIV) infection on lymphocyte numbers and T-cell turnover in different organs of rhesus monkeys.Leukocyte counts in cerebrospinal fluid and blood following firategrast treatment in subjects with relapsing forms of multiple sclerosis.Modulation of simian immunodeficiency virus neuropathology by dopaminergic drugs.Dopamine deficits and regulation of the cAMP second messenger system in brains of simian immunodeficiency virus-infected rhesus monkeys.Disruption of excitatory amino acid transporters in brains of SIV-infected rhesus macaques is associated with microglia activation.Susceptibility to Simian immunodeficiency virus ex vivo predicts outcome of a prime-boost vaccine after SIVmac239 challenge.Enrichment and persistence of virus-specific CTL in the brain of simian immunodeficiency virus-infected monkeys is associated with a unique cytokine environment.Homozygosity for a conserved Mhc class II DQ-DRB haplotype is associated with rapid disease progression in simian immunodeficiency virus-infected macaques: results from a prospective study.Regulation of glutathione and cell toxicity following exposure to neurotropic substances and human immunodeficiency virus-1 in vitro.The effect of simian immunodeficiency virus infection in vitro and in vivo on the cytokine production of isolated microglia and peripheral macrophages from rhesus monkey.
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