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Islet cell hyperexpression of HLA class I antigens: a defining feature in type 1 diabetesHistone deacetylases 1 and 3 but not 2 mediate cytokine-induced beta cell apoptosis in INS-1 cells and dispersed primary islets from rats and are differentially regulated in the islets of type 1 diabetic childrenSpatiotemporal Dynamics of Insulitis in Human Type 1 DiabetesGerminal centre frequency is decreased in pancreatic lymph nodes from individuals with recent-onset type 1 diabetes.Detection of a low-grade enteroviral infection in the islets of langerhans of living patients newly diagnosed with type 1 diabetes.Blood and islet phenotypes indicate immunological heterogeneity in type 1 diabetesAnalysis of islet inflammation in human type 1 diabetes.The prevalence of enteroviral capsid protein vp1 immunostaining in pancreatic islets in human type 1 diabetes.Differential cell autonomous responses determine the outcome of coxsackievirus infections in murine pancreatic α and β cellsDetection and localization of viral infection in the pancreas of patients with type 1 diabetes using short fluorescently-labelled oligonucleotide probes.Immunopathology of the human pancreas in type-I diabetes.Pancreatic pathology in type 1 diabetes mellitus.Islet inflammation in human type 1 diabetes mellitus.Wnt4 antagonises Wnt3a mediated increases in growth and glucose stimulated insulin secretion in the pancreatic beta-cell line, INS-1.An Isochemogenic Set of Inhibitors To Define the Therapeutic Potential of Histone Deacetylases in β-Cell Protection.Activity of thalidomide and lenalidomide in mantle cell lymphoma.Solution structure of prosurvival Mcl-1 and characterization of its binding by proapoptotic BH3-only ligands.miR, miR in the Cell, Does the Virus Control Them All?Relative sensitivity of immunohistochemistry, multiple reaction monitoring mass spectrometry, in situ hybridization and PCR to detect Coxsackievirus B1 in A549 cells.Differential Insulitic Profiles Determine the Extent of β-Cell Destruction and the Age at Onset of Type 1 Diabetes.Enteroviruses as causative agents in type 1 diabetes: loose ends or lost cause?Is type 1 diabetes "going viral"?Infection of human islets of Langerhans with two strains of Coxsackie B virus serotype 1: assessment of virus replication, degree of cell death and induction of genes involved in the innate immunity pathway.Evaluation of the fidelity of immunolabelling obtained with clone 5D8/1, a monoclonal antibody directed against the enteroviral capsid protein, VP1, in human pancreas.Erratum. Blood and Islet Phenotypes Indicate Immunological Heterogeneity in Type 1 Diabetes. Diabetes 2014;63:3835-3845Use of antisera directed against dsRNA to detect viral infections in formalin-fixed paraffin-embedded tissue.Immunohistochemical analysis of the relationship between islet cell proliferation and the production of the enteroviral capsid protein, VP1, in the islets of patients with recent-onset type 1 diabetes.The species origin of the cellular microenvironment influences markers of beta cell fate and function in EndoC-βH1 cells.A novel rat CVB1-VP1 monoclonal antibody 3A6 detects a broad range of enteroviruses.Single-agent lenalidomide in relapsed/refractory mantle cell lymphoma: results from a UK phase II study suggest activity and possible gender differences.Re-addressing the 2013 consensus guidelines for the diagnosis of insulitis in human type 1 diabetes: is change necessary?Molecular Pathways for Immune Recognition of Preproinsulin Signal Peptide in Type 1 Diabetes.Detection of enterovirus in the islet cells of patients with type 1 diabetes: what do we learn from immunohistochemistry? Reply to Hansson SF, Korsgren S, Pontén F et al [letter].The subcellular distribution of cyclin-D1 and cyclin-D3 within human islet cells varies according to the status of the pancreas donor.Evidence of increased islet cell proliferation in patients with recent-onset type 1 diabetes.Islet-associated macrophages in type 2 diabetes.The diagnosis of insulitis in human type 1 diabetes.C-Peptide Decline in Type 1 Diabetes Has Two Phases: An Initial Exponential Fall and a Subsequent Stable Phase.PDL1 is expressed in the islets of people with type 1 diabetes and is up-regulated by interferons-α and-γ via IRF1 inductionFifty years of pancreatic islet pathology in human type 1 diabetes: insights gained and progress made
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P50
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Sarah J. Richardson
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Sarah J. Richardson
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Sarah J. Richardson
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0000-0002-1160-6062