about
The circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarctionKnowledge gaps to understanding cardiac macrophage polarization following myocardial infarction.Citrate synthase is a novel in vivo matrix metalloproteinase-9 substrate that regulates mitochondrial function in the postmyocardial infarction left ventricle.Cardiac extracellular proteome profiling and membrane topology analysis using glycoproteomics.P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction.Plasma Glycoproteomics Reveals Sepsis Outcomes Linked to Distinct Proteins in Common Pathways.A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis.CD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling.Matrix metalloproteinases as input and output signals for post-myocardial infarction remodelingTemporal neutrophil polarization following myocardial infarctionCirculating Porphyromonas gingivalis lipopolysaccharide resets cardiac homeostasis in mice through a matrix metalloproteinase-9-dependent mechanism.Texas 3-step decellularization protocol: looking at the cardiac extracellular matrix.Myocardial Infarction Superimposed on Aging: MMP-9 Deletion Promotes M2 Macrophage Polarization.Matrix Metalloproteinases in Myocardial Infarction and Heart Failure.Defining the sham environment for post-myocardial infarction studies in miceIL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation.Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.Cardiac aging: Send in the vinculin reinforcements.Proteomic analysis of the cardiac extracellular matrix: clinical research applications.Understanding cardiac extracellular matrix remodeling to develop biomarkers of myocardial infarction outcomes.May the fibrosis be with you: Is discoidin domain receptor 2 the receptor we have been looking for?Mapping macrophage polarization over the myocardial infarction time continuum.The Mouse Heart Attack Research Tool 1.0 databaseLXR/RXR signaling and neutrophil phenotype following myocardial infarction classify sex differences in remodelingIron overload: what's TIMP-3 got to do with itRegulation of mitochondria function by natriuretic peptidesAdaptive immunity-driven inflammation and cardiovascular diseaseAn Offer We Cannot Refuse: Cell-Free DNA as a Novel Biomarker of Myocardial InfarctionCD8+ T-cells negatively regulate inflammation post-myocardial infarctionIdentifying the molecular and cellular signature of cardiac dilation following myocardial infarction
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description
researcher
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wetenschapper
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հետազոտող
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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Kristine Y DeLeon-Pennell
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P106
P31
P496
0000-0002-9647-6112