LCZ696, an angiotensin receptor-neprilysin inhibitor, improves cardiac function with the attenuation of fibrosis in heart failure with reduced ejection fraction in streptozotocin-induced diabetic mice.

LCZ696, an angiotensin receptor-neprilysin inhibitor, improves cardiac function with the attenuation of fibrosis in heart failure with reduced ejection fraction in streptozotocin-induced diabetic mice.

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http://www.wikidata.org/entity/Q53214797
Rescue of Heart Failure by Mitochondrial RecoveryFrom ARB to ARNI in Cardiovascular ControlUpdate on RAAS Modulation for the Treatment of Diabetic Cardiovascular DiseaseMechanisms of action of sacubitril/valsartan on cardiac remodeling: a systems biology approach.The angiotensin II type 1 receptor-neprilysin inhibitor LCZ696 blocked aldosterone synthesis in a human adrenocortical cell line.Angiotensin receptor neprilysin inhibitor LCZ696: pharmacology, pharmacokinetics and clinical development.Long-term neprilysin inhibition - implications for ARNIs.The evolution of natriuretic peptide augmentation in management of heart failure and the role of sacubitril/valsartan.CCAAT/enhancer-binding protein β overexpression alleviates myocardial remodelling by regulating angiotensin-converting enzyme-2 expression in diabetes.Intracardiac light catheter for rapid scanning transmural absorbance spectroscopy of perfused myocardium: measurement of myoglobin oxygenation and mitochondria redox state.LCZ696 (Sacubitril/valsartan) ameliorates oxidative stress, inflammation, fibrosis and improves renal function beyond angiotensin receptor blockade in CKD.Ejection fraction improvement and reverse remodeling achieved with Sacubitril/Valsartan in heart failure with reduced ejection fraction patients.LncRNAs: Proverbial Genomic "Junk" or Key Epigenetic Regulators During Cardiac Fibrosis in Diabetes?Combined Angiotensin Receptor-Neprilysin Inhibitors Improve Cardiac and Vascular Function Via Increased NO Bioavailability in Heart Failure.April 2016 at a glance. Focus on cardiac remodeling, biomarkers and treatment.MicroRNA-143 modulates the expression of Natriuretic Peptide Receptor 3 in cardiac cells.
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LCZ696, an angiotensin receptor-neprilysin inhibitor, improves cardiac function with the attenuation of fibrosis in heart failure with reduced ejection fraction in streptozotocin-induced diabetic mice.

Item
http://www.wikidata.org/entity/Q53214797
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
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2016年學術文章
@zh-hant
name
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@en
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@nl
type
label
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@en
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@nl
prefLabel
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@en
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@nl
P1433
P1476
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P2860
P304
P31
P356
P433
P478
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P1433
P1476
LCZ696, an angiotensin recepto ...... zotocin-induced diabetic mice.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921