Association of microRNA-34a overexpression with proliferation is cell type-dependent. - Wikidata - wikimedia - TriplyDB

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

http://www.wikidata.org/entity/Q53328782

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MicroRNA-34a modulates genes involved in cellular motility and oxidative phosphorylation in neural precursors derived from human umbilical cord mesenchymal stem cells Targeting CSCs in tumor microenvironment: the potential role of ROS-associated miRNAs in tumor aggressiveness Antioxidant Mechanisms and ROS-Related MicroRNAs in Cancer Stem Cells MiR-34a targeting of Notch ligand delta-like 1 impairs CD15+/CD133+ tumor-propagating cells and supports neural differentiation in medulloblastoma Cross-platform toxicogenomics for the prediction of non-genotoxic hepatocarcinogenesis in rat Linkage of microRNA and proteome-based profiling data sets: a perspective for the priorization of candidate biomarkers in renal cell carcinoma?MicroRNAs in cancer.An expression meta-analysis of predicted microRNA targets identifies a diagnostic signature for lung cancer.Identifying mRNA targets of microRNA dysregulated in cancer: with application to clear cell Renal Cell Carcinoma.The microRNA and messengerRNA profile of the RNA-induced silencing complex in human primary astrocyte and astrocytoma cells Low Expression of Circulating MicroRNA-34c is Associated with Poor Prognosis in Triple-Negative Breast Cancer Role of miR-34c microRNA in the late steps of spermatogenesis.MiR-34a expression has an effect for lower risk of metastasis and associates with expression patterns predicting clinical outcome in breast cancer.MicroRNA-34a suppresses invasion through downregulation of Notch1 and Jagged1 in cervical carcinoma and choriocarcinoma cells.MicroRNA-34 family expression in bovine gametes and preimplantation embryos miRNA expression profiles in myelodysplastic syndromes reveal Epstein-Barr virus miR-BART13 dysregulation.Circulating microRNAs as blood-based markers for patients with primary and metastatic breast cancer.A systematic analysis of the 3'UTR of HNF4A mRNA reveals an interplay of regulatory elements including miRNA target sites.Extracellular vesicles from bone marrow mesenchymal stem/stromal cells transport tumor regulatory microRNA, proteins, and metabolites.The Axl-Regulating Tumor Suppressor miR-34a Is Increased in ccRCC but Does Not Correlate with Axl mRNA or Axl Protein Levels.Circulating microRNAs - a new horizon in molecular diagnosis of breast cancer.MicroRNA profiling of clear cell renal cell cancer identifies a robust signature to define renal malignancy.MiR-34b is associated with clinical outcome in triple-negative breast cancer patients Unveiling of miRNA Expression Patterns in Purkinje Cells During Development.miR-34a regulates cell proliferation, morphology and function of newborn neurons resulting in improved behavioural outcomes Negative feedback regulation of AXL by miR-34a modulates apoptosis in lung cancer cells Marine algal natural products with anti-oxidative, anti-inflammatory, and anti-cancer properties Molecular signatures in urologic tumors.miRNA regulation of Sirtuin-1 expression in human astrocytoma.Small noncoding RNAs in cells transformed by human T-cell leukemia virus type 1: a role for a tRNA fragment as a primer for reverse transcriptase.MicroRNA dysregulation during chemical carcinogenesis.miRNA and TMPRSS2-ERG do not mind their own business in prostate cancer cells.Iron overload as a major targetable pathogenesis of asbestos-induced mesothelial carcinogenesis.MicroRNA-34a promotes genomic instability by a broad suppression of genome maintenance mechanisms downstream of the oncogene KSHV-vGPCR.MicroRNA expressions associated with progression of prostate cancer cells to antiandrogen therapy resistance.The prostate cancer-up-regulated Myc-associated zinc-finger protein (MAZ) modulates proliferation and metastasis through reciprocal regulation of androgen receptor.MicroRNA-34a inhibits human brain glioma cell growth by down-regulation of Notch1.Screening for circulating nucleic acids and caspase activity in the peripheral blood as potential diagnostic tools in lung cancer.Aging rather than sun exposure is a major determining factor for the density of miR-125b-positive epidermal stem cells in human skin.MicroRNA-34a: A Key Regulator in the Hallmarks of Renal Cell Carcinoma.

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Association of microRNA-34a overexpression with proliferation is cell type-dependent.

http://www.wikidata.org/entity/Q53328782

description

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2007年學術文章

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name

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@en

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@nl

type

label

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@en

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@nl

prefLabel

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@en

Association of microRNA-34a overexpression with proliferation is cell type-dependent.

@nl

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Association of microRNA-34a overexpression with proliferation is cell type-dependent

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Hiroshi Masutani

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Khokon Kumar Dutta

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Masanori Takehashi

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Minako Yoshihara

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Shinya Akatsuka

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Takuji Yamada

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P2860

Human microRNA genes are frequently located at fragile sites and genomic regions involved in cancers

Real-time expression profiling of microRNA precursors in human cancer cell lines.

MicroRNA gene expression deregulation in human breast cancer

MicroRNAs: genomics, biogenesis, mechanism, and function

Temporal regulation of microRNA expression in Drosophila melanogaster mediated by hormonal signals and broad-Complex gene activity

Embryonic stem cell-specific MicroRNAs

Depletion of human micro-RNA miR-125b reveals that it is critical for the proliferation of differentiated cells but not for the down-regulation of putative targets during differentiation

MicroRNA-34a functions as a potential tumor suppressor by inducing apoptosis in neuroblastoma cells

Rbx1, a component of the VHL tumor suppressor complex and SCF ubiquitin ligase

Two distinct mechanisms for loss of thioredoxin-binding protein-2 in oxidative stress-induced renal carcinogenesis

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