Nickel compounds render anti-apoptotic effect to human bronchial epithelial Beas-2B cells by induction of cyclooxygenase-2 through an IKKbeta/p65-dependent and IKKalpha- and p50-independent pathway.
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The effect of environmental chemicals on the tumor microenvironmentHexavalent chromium Cr(VI) up-regulates COX-2 expression through an NFκB/c-Jun/AP-1-dependent pathwayNickel alterations of TLR2-dependent chemokine profiles in lung fibroblasts are mediated by COX-2p27 suppresses cyclooxygenase-2 expression by inhibiting p38β and p38δ-mediated CREB phosphorylation upon arsenite exposureCauses of genome instability: the effect of low dose chemical exposures in modern societyAnti-apoptotic proteins and catalase-dependent apoptosis resistance in nickel chloride-transformed human lung epithelial cellsAssessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge aheadUpregulation of SQSTM1/p62 contributes to nickel-induced malignant transformation of human bronchial epithelial cellsLoss of p27 upregulates MnSOD in a STAT3-dependent manner, disrupts intracellular redox activity and enhances cell migration.Epigenetic dysregulation by nickel through repressive chromatin domain disruption.GADD45α induction by nickel negatively regulates JNKs/p38 activation via promoting PP2Cα expression.p53 activation by Ni(II) is a HIF-1α independent response causing caspases 9/3-mediated apoptosis in human lung cells.Nickel compounds induce apoptosis in human bronchial epithelial Beas-2B cells by activation of c-Myc through ERK pathwayA cross-talk between NFAT and NF-κB pathways is crucial for nickel-induced COX-2 expression in Beas-2B cellsProtective Effects of Gallic Acid Against NiSO4-Induced Toxicity Through Down-Regulation of the Ras/ERK Signaling Pathway in Beas-2B CellsEffects of nickel on cyclin expression, cell cycle progression and cell proliferation in human pulmonary cells.Nickel-smelting fumes increased the expression of HIF-1α through PI3K/ERK pathway in NIH/3T3 cells.Toxicological effects of major environmental pollutants: an overview.Hydrogen peroxide/ATR-Chk2 activation mediates p53 protein stabilization and anti-cancer activity of cheliensisin A in human cancer cells.Mucin 1 (MUC1) signalling contributes to increase the resistance to cell death in human bronchial epithelial cells exposed to nickel acetate.Cheliensisin A inhibits EGF-induced cell transformation with stabilization of p53 protein via a hydrogen peroxide/Chk1-dependent axis.Bid mediates anti-apoptotic COX-2 induction through the IKKbeta/NFkappaB pathway due to 5-MCDE exposure.
P2860
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P2860
Nickel compounds render anti-apoptotic effect to human bronchial epithelial Beas-2B cells by induction of cyclooxygenase-2 through an IKKbeta/p65-dependent and IKKalpha- and p50-independent pathway.
description
2006 nî lūn-bûn
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2006年の論文
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name
Nickel compounds render anti-a ...... - and p50-independent pathway.
@en
Nickel compounds render anti-a ...... - and p50-independent pathway.
@nl
type
label
Nickel compounds render anti-a ...... - and p50-independent pathway.
@en
Nickel compounds render anti-a ...... - and p50-independent pathway.
@nl
prefLabel
Nickel compounds render anti-a ...... - and p50-independent pathway.
@en
Nickel compounds render anti-a ...... - and p50-independent pathway.
@nl
P2093
P2860
P356
P1476
Nickel compounds render anti-a ...... - and p50-independent pathway.
@en
P2093
Caifang Xue
Dongyun Zhang
J Andrés Meléndez
Jingxia Li
Weiming Ouyang
Xinhai Zhang
P2860
P304
39022-39032
P356
10.1074/JBC.M604798200
P407
P577
2006-09-18T00:00:00Z