Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
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Induced Pluripotent Stem Cells: Generation Strategy and Epigenetic Mystery behind ReprogrammingSignaling pathways in induced naïve pluripotencyAkt3 is responsible for the survival and proliferation of embryonic stem cellsGenetic and epigenetic analysis of putative breast cancer stem cell models.G-CSF Promotes Neuroblastoma Tumorigenicity and Metastasis via STAT3-Dependent Cancer Stem Cell Activation.Contributions of the Epidermal Growth Factor Receptor to Acquisition of Platinum Resistance in Ovarian Cancer Cells.Role of STAT3 in lung cancer.JAK/STAT3 signaling pathway mediates endothelial-like differentiation of immature dendritic cells.Establishment of leukemia inhibitory factor (LIF)-independent iPS cells with potentiated Oct4Expression of the miR-302/367 cluster in glioblastoma cells suppresses tumorigenic gene expression patterns and abolishes transformation related phenotypes.Stat3 promotes mitochondrial transcription and oxidative respiration during maintenance and induction of naive pluripotency.STAT3 in the systemic inflammation of cancer cachexia.Targeting cancer stem cells: emerging role of Nanog transcription factorIdentification of the missing pluripotency mediator downstream of leukaemia inhibitory factor.JAK-STAT3 and somatic cell reprogramming.Secretome from senescent melanoma engages the STAT3 pathway to favor reprogramming of naive melanoma towards a tumor-initiating cell phenotype.NANOG: a promising target for digestive malignant tumorsA critical role for p38MAPK signalling pathway during reprogramming of human fibroblasts to iPSCs.Signal transducer and activator of transcription (STAT)-3 regulates microRNA gene expression in chronic lymphocytic leukemia cells.Knockdown of Brm and Baf170, Components of Chromatin Remodeling Complex, Facilitates Reprogramming of Somatic Cells.Stat3 inhibits PTPN13 expression in squamous cell lung carcinoma through recruitment of HDAC5.Tumor heterogeneity, clonal evolution, and therapy resistance: an opportunity for multitargeting therapy.Synergistic inhibition of tumor growth by combination treatment with drugs against different subpopulations of glioblastoma cells.LIF-activated Jak signaling determines Esrrb expression during late-stage reprogramming.JAK/STAT3 regulated global gene expression dynamics during late-stage reprogramming process.Ciliary neurotrophic factor promotes the activation of corneal epithelial stem/progenitor cells and accelerates corneal epithelial wound healing.siRNA Library Screening Identifies a Druggable Immune-Signature Driving Esophageal Adenocarcinoma Cell Growth.
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P2860
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@en
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@nl
type
label
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@en
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@nl
prefLabel
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@en
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@nl
P2093
P2860
P356
P1433
P1476
Jak/Stat3 signaling promotes somatic cell reprogramming by epigenetic regulation.
@en
P2093
Chih-Jen Lin
Joonghong Park
Mark G Carter
Sharon Kish
Tomokazu Amano
Xiuchun Cindy Tian
Yinghong Ma
P2860
P304
P356
10.1002/STEM.1225
P577
2012-12-01T00:00:00Z