Jieduquyuziyin prescription suppresses IL-17 production and Th17 activity in MRL/lpr mice by inhibiting expression of Ca(2+)/calmodulin-dependent protein kinase-4.

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Jieduquyuziyin prescription suppresses IL-17 production and Th17 activity in MRL/lpr mice by inhibiting expression of Ca(2+)/calmodulin-dependent protein kinase-4.

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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Jieduquyuziyin prescription su ...... by inhibiting expression of Ca

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Jieduquyuziyin prescription su ...... in-dependent protein kinase-4.

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type

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label

Jieduquyuziyin prescription su ...... by inhibiting expression of Ca

@nl

Jieduquyuziyin prescription su ...... in-dependent protein kinase-4.

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prefLabel

Jieduquyuziyin prescription su ...... by inhibiting expression of Ca

@nl

Jieduquyuziyin prescription su ...... in-dependent protein kinase-4.

@en

P1476

Jieduquyuziyin prescription su ...... in-dependent protein kinase-4.

@en

P2093

Bingjie Shui

http://www.w3.org/2001/XMLSchema#string

Chengping Wen

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Weibiao Xia

http://www.w3.org/2001/XMLSchema#string

Xinghong Ding

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P2860

Calcium/calmodulin-dependent kinase IV in immune and inflammatory responses: novel routes for an ancient traveller

IL-17 and Th17 Cells

Rapid resolution liquid chromatography coupled with quadrupole time-of-flight mass spectrometry-based metabolomics approach to study the effects of jieduquyuziyin prescription on systemic lupus erythematosus

T-helper 17 cell cytokines and interferon type I: partners in crime in systemic lupus erythematosus?

Increased expression of costimulatory markers CD134 and CD80 on interleukin-17 producing T cells in patients with systemic lupus erythematosus.

Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.

Cutting edge: Calcium/Calmodulin-dependent protein kinase type IV is essential for mesangial cell proliferation and lupus nephritis.

cAMP-responsive element modulator (CREM)α protein induces interleukin 17A expression and mediates epigenetic alterations at the interleukin-17A gene locus in patients with systemic lupus erythematosus

cAMP-responsive element modulator α (CREMα) suppresses IL-17F protein expression in T lymphocytes from patients with systemic lupus erythematosus (SLE).

Calcium/calmodulin-dependent protein kinase IV suppresses IL-2 production and regulatory T cell activity in lupus

P2888

s11418-015-0900-1

P304

349-357

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scholarly article

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10.1007/S11418-015-0900-1

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-03-28T00:00:00Z

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P698

25821132

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