Regional myocardial ischemia-induced activation of MAPKs is associated with subcellular redistribution of caveolin and cholesterol.
about
Cardiac-specific overexpression of caveolin-3 induces endogenous cardiac protection by mimicking ischemic preconditioningPopeye domain containing 1 (Popdc1/Bves) is a caveolae-associated protein involved in ischemia toleranceGene expression profile of rat left ventricles reveals persisting changes following chronic mild exercise protocol: implications for cardioprotection.Cardiac function is not significantly diminished in hearts isolated from young caveolin-1 knockout mice.Signaling epicenters: the role of caveolae and caveolins in volatile anesthetic induced cardiac protection.Role of caveolae in cardiac protection.Caveolae as organizers of pharmacologically relevant signal transduction molecules.Caveolins in cardioprotection - translatability and mechanismsEnhanced myogenic constriction of mesenteric artery in heart failure relates to decreased smooth muscle cell caveolae numbers and altered AT1- and epidermal growth factor-receptor function.Cholesterol Depletion Alters Cardiomyocyte Subcellular Signaling and Increases Contractility.TIR/BB-loop mimetic AS-1 attenuates cardiac ischemia/reperfusion injury via a caveolae and caveolin-3-dependent mechanism.Lipid raft in cardiac health and disease.Caveolae, caveolin, and cavins: potential targets for the treatment of cardiac disease.Caveolae: molecular insights and therapeutic targets for stroke.Caveolin-1/-3: therapeutic targets for myocardial ischemia/reperfusion injury.Sarcolemmal dependence of cardiac protection and stress-resistance: roles in aged or diseased hearts.Cell proliferation in in vivo-like three-dimensional cell culture is regulated by sequestration of ERK1/2 to lipid rafts.Caveolins and cavins in the trafficking, maturation, and degradation of caveolae: implications for cell physiology.Delta Opioid Receptors and Cardioprotection.The deubiquitinating enzyme USP17 blocks N-Ras membrane trafficking and activation but leaves K-Ras unaffected.MEK1-induced physiological hypertrophy inhibits chronic post-myocardial infarction remodeling in mice.Oxidative stress and adenosine A1 receptor activation differentially modulate subcellular cardiomyocyte MAPKs.Pharmacological activation of PPARγ inhibits hypoxia-induced proliferation through a caveolin-1 targeted and dependent mechanism in PASMCs.Caveolins as regulators of stress adaptation.Helium alters the cytoskeleton and decreases permeability in endothelial cells cultured in vitro through a pathway involving Caveolin-1.
P2860
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P2860
Regional myocardial ischemia-induced activation of MAPKs is associated with subcellular redistribution of caveolin and cholesterol.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
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2006年學術文章
@zh-hant
name
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@en
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@nl
type
label
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@en
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@nl
prefLabel
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@en
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@nl
P2093
P2860
P1476
Regional myocardial ischemia-i ...... n of caveolin and cholesterol.
@en
P2093
Adam C Locklar
Cherry Ballard-Croft
Gentian Kristo
Robert D Lasley
P2860
P304
P356
10.1152/AJPHEART.01354.2005
P577
2006-03-24T00:00:00Z