HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response. - Wikidata - wikimedia - TriplyDB

HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response.

HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response.

http://www.wikidata.org/entity/Q53663069

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Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Lighting the fires within: the cell biology of autoinflammatory diseases HLA-B27 misfolding and ankylosing spondylitis Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyond The role of intracellular organisms in the pathogenesis of inflammatory arthritis From HLA-B27 to spondyloarthritis: a journey through the ER.Role of endoplasmic reticulum stress in rheumatoid arthritis pathogenesis.Toll-like receptor-mediated IRE1α activation as a therapeutic target for inflammatory arthritis Inflammatory bowel disease Spondyloarthritis: Matrix Metalloproteinasesas Biomarkers of Pathogenesis and Response to Tumor Necrosis Factor (TNF) Inhibitors.ERAP1 reduces accumulation of aberrant and disulfide-linked forms of HLA-B27 on the cell surface Gene expression analysis of macrophages derived from ankylosing spondylitis patients reveals interferon-gamma dysregulation HLA-B27 misfolding and the unfolded protein response augment interleukin-23 production and are associated with Th17 activation in transgenic rats.XBP-1 couples endoplasmic reticulum stress to augmented IFN-beta induction via a cis-acting enhancer in macrophages.Endoplasmic reticulum stress-induced transcription factor, CHOP, is crucial for dendritic cell IL-23 expression Genes and environment: how will our concepts on the pathophysiology of IBD develop in the future?Phosphorylation of STAT-1 serine 727 is prolonged in HLA-B27-expressing human monocytic cells.Functionally distinct ERAP1 allotype combinations distinguish individuals with Ankylosing Spondylitis.Pathogenicity of Misfolded and Dimeric HLA-B27 Molecules Targeted delivery of an antigenic peptide to the endoplasmic reticulum: application for development of a peptide therapy for ankylosing spondylitis.ZAP-70 genotype disrupts the relationship between microbiota and host, leading to spondyloarthritis and ileitis in SKG mice.Ankylosing spondylitis macrophage production of higher levels of interleukin-23 in response to lipopolysaccharide without induction of a significant unfolded protein response.Major histocompatibility complex associations of ankylosing spondylitis are complex and involve further epistasis with ERAP1.Inflammatory bowel disease associated arthropathy Interactions of the innate and adaptive arms of the immune system in the pathogenesis of spondyloarthritis Altered PKR Signalling and C / EBPβ Expression is Associated with HLA-B27 Expression in Monocytic Cells.The unfolded protein response and its role in intestinal homeostasis and inflammation.Introduction: the unfolded protein response's role in disease pathophysiology.Autophagy, microbial sensing, endoplasmic reticulum stress, and epithelial function in inflammatory bowel disease Sulfasalazine Treatment Suppresses the Formation of HLA-B27 Heavy Chain Homodimer in Patients with Ankylosing Spondylitis.Mechanisms of Disease: the immunopathogenesis of spondyloarthropathies.The unfolded protein response and gastrointestinal disease Peptides: the cornerstone of HLA-B27 biology and pathogenetic role in spondyloarthritis.HLA-B27 misfolding and spondyloarthropathies.HLA-B35 upregulates endothelin-1 and downregulates endothelial nitric oxide synthase via endoplasmic reticulum stress response in endothelial cells.Joint expedition: linking gut inflammation to arthritis.HLA-B27 alters the response to tumor necrosis factor α and promotes osteoclastogenesis in bone marrow monocytes from HLA-B27-transgenic rats.Endoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease.Evidence that autophagy, but not the unfolded protein response, regulates the expression of IL-23 in the gut of patients with ankylosing spondylitis and subclinical gut inflammation Progress in spondylarthritis. Immunopathogenesis of spondyloarthritis: which cells drive disease?

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P2860

HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response.

http://www.wikidata.org/entity/Q53663069

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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name

HLA-B27 misfolding in transgen ...... the unfolded protein response.

@en

HLA-B27 misfolding in transgen ...... the unfolded protein response.

@nl

type

label

HLA-B27 misfolding in transgen ...... the unfolded protein response.

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HLA-B27 misfolding in transgen ...... the unfolded protein response.

@nl

prefLabel

HLA-B27 misfolding in transgen ...... the unfolded protein response.

@en

HLA-B27 misfolding in transgen ...... the unfolded protein response.

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JIMMUNOL.175.4.2438

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Journal of Immunology

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HLA-B27 misfolding in transgen ...... the unfolded protein response.

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Dawn P Sowders

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Jaclyn R Brandewie

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Joel D Taurog

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Judith A Smith

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Matthew J Turner

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Monica L DeLay

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Rajashree Mohapatra

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Robert A Colbert

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Shuzhen Bai

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10.4049/JIMMUNOL.175.4.2438

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