Loss of the CONSTITUTIVE PHOTOMORPHOGENIC9 signalosome subunit 5 is sufficient to cause the cop/det/fus mutant phenotype in Arabidopsis.
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The NEDD8 modification pathway in plantsTargeted silencing of Jab1/Csn5 in human cells downregulates SCF activity through reduction of F-box protein levels.Downregulation of COP9 signalosome subunits differentially affects the CSN complex and target protein stabilityThe Arabidopsis Cop9 signalosome subunit 4 (CNS4) is involved in adventitious root formationDrosophila COP9 signalosome subunit 7 interacts with multiple genomic loci to regulate development.Regulation of the Nrf2-Keap1 antioxidant response by the ubiquitin proteasome system: an insight into cullin-ring ubiquitin ligases.Transcriptome comparison and gene coexpression network analysis provide a systems view of citrus response to 'Candidatus Liberibacter asiaticus' infection.Arabidopsis CSN5B interacts with VTC1 and modulates ascorbic acid synthesis.Composition, roles, and regulation of cullin-based ubiquitin e3 ligases.The eta7/csn3-3 auxin response mutant of Arabidopsis defines a novel function for the CSN3 subunit of the COP9 signalosome.Revisiting the COP9 signalosome as a transcriptional regulator.An eight-subunit COP9 signalosome with an intact JAMM motif is required for fungal fruit body formation.The organization of a CSN5-containing subcomplex of the COP9 signalosome.Genomic analysis of COP9 signalosome function in Drosophila melanogaster reveals a role in temporal regulation of gene expression.Characterization of the VIER F-BOX PROTEINE genes from Arabidopsis reveals their importance for plant growth and development.The Arabidopsis calmodulin-like protein, CML39, functions during early seedling establishment.Diversity of COP9 signalosome structures and functional consequences.Molecular analysis of T-DNA insertion mutants identified putative regulatory elements in the AtTERT gene.D27E mutation of VTC1 impairs the interaction with CSN5B and enhances ascorbic acid biosynthesis and seedling growth in Arabidopsis.The Clubroot Pathogen (Plasmodiophora brassicae) Influences Auxin Signaling to Regulate Auxin Homeostasis in Arabidopsis.The COP9 signalosome regulates Skp2 levels and proliferation of human cells.COP9 signalosome- and 26S proteasome-dependent regulation of SCFTIR1 accumulation in Arabidopsis.The COP9 signalosome controls jasmonic acid synthesis and plant responses to herbivory and pathogens.Novel aspects of COP9 signalosome functions revealed through analysis of hypomorphic csn mutants.Mutation of E1-CONJUGATING ENZYME-RELATED1 decreases RELATED TO UBIQUITIN conjugation and alters auxin response and development.Genetical Genomics Reveals Large Scale Genotype-By-Environment Interactions in Arabidopsis thaliana.incurvata13, a novel allele of AUXIN RESISTANT6, reveals a specific role for auxin and the SCF complex in Arabidopsis embryogenesis, vascular specification, and leaf flatness.Neddylation and deneddylation regulate Cul1 and Cul3 protein accumulation.MAP kinases associate with high molecular weight multiprotein complexes.LucTrap vectors are tools to generate luciferase fusions for the quantification of transcript and protein abundance in vivo.DENEDDYLASE1 Protein Counters Automodification of Neddylating Enzymes to Maintain NEDD8 Protein Homeostasis in Arabidopsis.Plant Deubiquitinases and Their Role in the Control of Gene Expression Through Modification of Histones.The COP9 Signalosome regulates seed germination by facilitating protein degradation of RGL2 and ABI5.The COP9 signalosome: its regulation of cullin-based E3 ubiquitin ligases and role in photomorphogenesis.Auxin responses in mutants of the Arabidopsis CONSTITUTIVE PHOTOMORPHOGENIC9 signalosome.Deneddylase 1 regulates deneddylase activity of the Cop9 signalosome in Drosophila melanogaster.COP9 signalosome subunit 5A affects phenylpropanoid metabolism, trichome formation and transcription of key genes of a regulatory tri-protein complex in Arabidopsis.DENEDDYLASE1 deconjugates NEDD8 from non-cullin protein substrates in Arabidopsis thaliana
P2860
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P2860
Loss of the CONSTITUTIVE PHOTOMORPHOGENIC9 signalosome subunit 5 is sufficient to cause the cop/det/fus mutant phenotype in Arabidopsis.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
@zh-hans
2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@en
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@nl
type
label
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@en
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@nl
prefLabel
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@en
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@nl
P2860
P356
P1433
P1476
Loss of the CONSTITUTIVE PHOTO ...... tant phenotype in Arabidopsis.
@en
P2093
Carola Kuhnle
Esther M N Dohmann
P2860
P304
P356
10.1105/TPC.105.032870
P577
2005-05-27T00:00:00Z