Cigarette smoke disrupts VEGF165-VEGFR-2 receptor signaling complex in rat lungs and patients with COPD: morphological impact of VEGFR-2 inhibition.
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COPD immunopathologyPhysiological impairment in mild COPDNeuropilins: expression and roles in the epitheliumAnimal models of chronic obstructive pulmonary diseaseEndothelial microparticles in mild chronic obstructive pulmonary disease and emphysema. The Multi-Ethnic Study of Atherosclerosis Chronic Obstructive Pulmonary Disease studyBioactive lipid profiling reveals drug target engagement of a soluble epoxide hydrolase inhibitor in a murine model of tobacco smoke exposureCigarette smoke regulates VEGFR2-mediated survival signaling in rat lungsRegulation of Human MUC7 Mucin Gene Expression by Cigarette Smoke Extract or Cigarette Smoke and Pseudomonas aeruginosa Lipopolysaccharide in Human Airway Epithelial Cells and in MUC7 Transgenic Mice.p53 mediates cigarette smoke-induced apoptosis of pulmonary endothelial cells: inhibitory effects of macrophage migration inhibitor factorSerum levels of vascular endothelial growth factor in chronic obstructive pulmonary diseaseCigarette smoke and α,β-unsaturated aldehydes elicit VEGF release through the p38 MAPK pathway in human airway smooth muscle cells and lung fibroblasts.Oxidative DNA damage in lung tissue from patients with COPD is clustered in functionally significant sequences.Oxidative stress induced lung cancer and COPD: opportunities for epigenetic therapy.Targeted deletion of Jun/AP-1 in alveolar epithelial cells causes progressive emphysema and worsens cigarette smoke-induced lung inflammationStress responses affecting homeostasis of the alveolar capillary unit.VEGFR-2 inhibition augments cigarette smoke-induced oxidative stress and inflammatory responses leading to endothelial dysfunctionGenetic ablation of NADPH oxidase enhances susceptibility to cigarette smoke-induced lung inflammation and emphysema in mice.Role of lung maintenance program in the heterogeneity of lung destruction in emphysema.Increased expression of placenta growth factor in COPDFunctional significance of apoptosis in chronic obstructive pulmonary disease.Cellular and molecular mechanisms of cigarette smoke-induced lung damage and prevention by vitamin C.Future therapeutic treatment of COPD: struggle between oxidants and cytokinesNeutrophil elastase cleaves VEGF to generate a VEGF fragment with altered activity.Prevention of elastase-induced emphysema in placenta growth factor knock-out mice.Alveolar epithelial and endothelial cell apoptosis in emphysema: what we know and what we need to know.Therapeutic potential of growth factors in pulmonary emphysematous condition.Physiological and pathological angiogenesis in the adult pulmonary circulation.Pathophysiology of tobacco smoke exposure: recent insights from comparative and redox proteomics.Semaphorins and Their Roles in Airway Biology: Potential as Therapeutic Targets.Bridging Lung Development with Chronic Obstructive Pulmonary Disease. Relevance of Developmental Pathways in Chronic Obstructive Pulmonary Disease Pathogenesis.Cigarette smoke extract induces placental growth factor release from human bronchial epithelial cells via ROS/MAPK (ERK-1/2)/Egr-1 axis.Experimental animal models for COPD: a methodological review.Azithromycin attenuates cigarette smoke extract-induced oxidative stress injury in human alveolar epithelial cells.Predictors of long-term compensatory response of pulmonary function following major lung resection for non-small cell lung cancer.The potential role of endothelial dysfunction and platelet activation in the development of thrombotic risk in COPD patients.Disruption of p21 attenuates lung inflammation induced by cigarette smoke, LPS, and fMLP in mice.Pathobiology of cigarette smoke-induced chronic obstructive pulmonary disease.Neuropilin-1 Is Expressed on Lymphoid Tissue Residing LTi-like Group 3 Innate Lymphoid Cells and Associated with Ectopic Lymphoid AggregatesCigarette-smoke-induced oxidative/nitrosative stress impairs VEGF- and fluid-shear-stress-mediated signaling in endothelial cells.Mitochondrial E3 Ubiquitin Protein Ligase 1 Mediates Cigarette Smoke-Induced Endothelial Cell Death and Dysfunction.
P2860
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P2860
Cigarette smoke disrupts VEGF165-VEGFR-2 receptor signaling complex in rat lungs and patients with COPD: morphological impact of VEGFR-2 inhibition.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@en
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@nl
type
label
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@en
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@nl
prefLabel
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@en
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@nl
P2093
P1476
Cigarette smoke disrupts VEGF1 ...... impact of VEGFR-2 inhibition.
@en
P2093
Christopher S Stevenson
Irfan Rahman
John A Marwick
June Giddings
Keith Butler
Paul A Kirkham
P304
P356
10.1152/AJPLUNG.00116.2005
P577
2005-12-16T00:00:00Z