Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats. - Wikidata - wikimedia - TriplyDB

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

http://www.wikidata.org/entity/Q54029205

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17Beta-estradiol decreases hypoxic induction of erythropoietin gene expression Novel role of endothelial BKCa channels in altered vasoreactivity following hypoxia Heme oxygenase metabolites inhibit tubuloglomerular feedback in vivo Antioxidant and vasodilatory effects of heme oxygenase on mesenteric vasoreactivity following chronic hypoxia.Inhibition of heme oxygenase augments tubular sodium reabsorption.Role of caveolin-1 in endothelial BKCa channel regulation of vasoreactivity.Dependence of acetylcholine and ADP dilation of pial arterioles on heme oxygenase after transfusion of cell-free polymeric hemoglobin.Renal heme oxygenase-1 induction with hemin augments renal hemodynamics, renal autoregulation, and excretory function Regulation of endothelial BK channels by heme oxygenase-derived carbon monoxide and caveolin-1.Heme oxygenase metabolites inhibit tubuloglomerular feedback (TGF)Heme oxygenase induction attenuates afferent arteriolar autoregulatory responses Increased nitric oxide production following chronic hypoxia contributes to attenuated systemic vasoconstriction.Correlation of HO-1 expression with onset and reversal of hypoxia-induced vasoconstrictor hyporeactivity.Homogeneous segmental profile of carbon monoxide-mediated pulmonary vasodilation in rats.Role of CO in attenuated vasoconstrictor reactivity of mesenteric resistance arteries after chronic hypoxia.48-h Hypoxic exposure results in endothelium-dependent systemic vascular smooth muscle cell hyperpolarization.Endogenous carbon monoxide is an endothelial-derived vasodilator factor in the mesenteric circulation.Heme oxygenase-mediated vasodilation involves vascular smooth muscle cell hyperpolarization.Interaction between endothelial heme oxygenase-2 and endothelin-1 in altered aortic reactivity after hypoxia in rats.Chronic intermittent hypoxia alters NE reactivity and mechanics of skeletal muscle resistance arteries.Endothelial-dependent dilation following chronic hypoxia involves TRPV4-mediated activation of endothelial BK channels.

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P2860

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

http://www.wikidata.org/entity/Q54029205

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2000 nî lūn-bûn

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2000年の論文

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年学术文章

@zh-my

2000年学术文章

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2000年學術文章

@yue

2000年學術文章

@zh-hant

name

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@en

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@nl

type

label

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@en

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@nl

prefLabel

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@en

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@nl

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AJPHEART.2000.279.6.H2908

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

Renal vasodilatory influence of endogenous carbon monoxide in chronically hypoxic rats.

@en

P2093

B R Walker

http://www.w3.org/2001/XMLSchema#string

T L O'Donaughy

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P2860

Effects of chronic hypoxia and altered hemodynamics on endothelial nitric oxide synthase expression in the adult rat lung.

Heme oxygenase is the major 32-kDa stress protein induced in human skin fibroblasts by UVA radiation, hydrogen peroxide, and sodium arsenite.

Smooth muscle cell-derived carbon monoxide is a regulator of vascular cGMP.

Effect of chronic hypoxia on adrenoceptor responses of ovine foetal umbilical vessels

Carbon monoxide is a major contributor to the regulation of vascular tone in aortas expressing high levels of haeme oxygenase-1

Isolation and characterization of a cDNA from the rat brain that encodes hemoprotein heme oxygenase-3.

NO-mediated activation of heme oxygenase: endogenous cytoprotection against oxidative stress to endothelium.

The heme oxygenase system: a regulator of second messenger gases.

Increased endothelium-derived NO in hypertensive pulmonary circulation of chronically hypoxic rats.

Activation of soluble guanylate cyclase by carbon monoxide and inhibition by superoxide anion.

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6

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carbon monoxide