Crosstalk between Akt signaling and cold shock proteins in mediating invasive cell phenotypes.

about

Crosstalk between Akt signaling and cold shock proteins in mediating invasive cell phenotypes.

Item

http://www.wikidata.org/entity/Q54112804

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh

2018年學術文章

@zh-hant

name

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@en

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@nl

type

Item

label

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@en

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@nl

prefLabel

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@en

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@nl

P1476

Crosstalk between Akt signalin ...... ting invasive cell phenotypes.

@en

P2093

Anja Bernhardt

http://www.w3.org/2001/XMLSchema#string

Berend Isermann

http://www.w3.org/2001/XMLSchema#string

Burkhard Jandrig

http://www.w3.org/2001/XMLSchema#string

Daniel Schindele

http://www.w3.org/2001/XMLSchema#string

Jonathan A Lindquist

http://www.w3.org/2001/XMLSchema#string

Martin Schostak

http://www.w3.org/2001/XMLSchema#string

Peter R Mertens

http://www.w3.org/2001/XMLSchema#string

Raphael Hohlfeld

http://www.w3.org/2001/XMLSchema#string

Sabine Brandt

http://www.w3.org/2001/XMLSchema#string

Xenia Gorny

http://www.w3.org/2001/XMLSchema#string

P2860

The Hallmarks of Cancer

Hallmarks of Cancer: The Next Generation

C1QBP negatively regulates the activation of oncoprotein YBX1 in the renal cell carcinoma as revealed by interactomics analysis

A host YB-1 ribonucleoprotein complex is hijacked by hepatitis C virus for the control of NS3-dependent particle production

Cold shock domain family members YB-1 and MSY4 share essential functions during murine embryogenesis

Nuclear detection of Y-box protein-1 (YB-1) closely associates with progesterone receptor negativity and is a strong adverse survival factor in human breast cancer.

Inhibition of protein synthesis by Y box-binding protein 1 blocks oncogenic cell transformation.

Inhibition of BET bromodomain targets genetically diverse glioblastoma

YB-1 transforms human mammary epithelial cells through chromatin remodeling leading to the development of basal-like breast cancer

The pleiotropic functions of the Y-box-binding protein, YB-1.

P304

19039-19049

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.18632/ONCOTARGET.24886

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-04-10T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29721182

http://www.w3.org/2001/XMLSchema#string

P932

5922376

http://www.w3.org/2001/XMLSchema#string