Endogenous ornithine decarboxylase/polyamine system mediated the antagonist role of insulin/PEG-CMCS preconditioning against heart ischemia/reperfusion injury in diabetes mellitus.

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Endogenous ornithine decarboxylase/polyamine system mediated the antagonist role of insulin/PEG-CMCS preconditioning against heart ischemia/reperfusion injury in diabetes mellitus.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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name

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

@en

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

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type

Item

label

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

@en

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

@nl

prefLabel

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

@en

Endogenous ornithine decarboxy ...... n injury in diabetes mellitus.

@nl

P1476

Endogenous ornithine decarboxy ...... on injury in diabetes mellitus

@en

P2093

Bing Yan

http://www.w3.org/2001/XMLSchema#string

Shiwei Ruan

http://www.w3.org/2001/XMLSchema#string

Shuyu Yang

http://www.w3.org/2001/XMLSchema#string

Suhuan Liu

http://www.w3.org/2001/XMLSchema#string

Xuejun Li

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P2860

Preparation of exenatide-loaded linear poly(ethylene glycol)-brush poly(l-lysine) block copolymer: potential implications on diabetic nephropathy.

Myocardial reperfusion injury.

Reperfusion therapy with low-dose insulin or insulin-like growth factor 2; myocardial function and infarct size in a porcine model of ischaemia and reperfusion.

Effects of polyamines on mitochondrial Ca(2+) transport.

Signal transduction pathways linking polyamines to apoptosis.

Mammalian polyamine catabolism: a therapeutic target, a pathological problem, or both?

The effect of insulin-loaded linear poly(ethylene glycol)-brush-like poly(l-lysine) block copolymer on renal ischemia/reperfusion-induced lung injury through downregulating hypoxia-inducible factor.

Induction of ornithine decarboxylase activity by insulin and growth factors is mediated by amino acids.

Simvastatin nanoparticles attenuated intestinal ischemia/reperfusion injury by downregulating BMP4/COX-2 pathway in rats.

Different prooxidant levels stimulate growth, trigger apoptosis, or produce necrosis of insulin-secreting RINm5F cells. The role of intracellular polyamines.

P304

2507-2520

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P31

scholarly article

P356

10.2147/IJN.S160848

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P407

English

P478

http://www.w3.org/2001/XMLSchema#string

P50

Fei Tong

P577

2018-04-23T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29719397

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P921

reperfusion injury

P932

5922236

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