Neutral endopeptidase (NEP) and its role in pathological pulmonary change with inhalation exposure to JP-8 jet fuel. - Wikidata - wikimedia - TriplyDB

Neutral endopeptidase (NEP) and its role in pathological pulmonary change with inhalation exposure to JP-8 jet fuel.

Neutral endopeptidase (NEP) and its role in pathological pulmonary change with inhalation exposure to JP-8 jet fuel.

http://www.wikidata.org/entity/Q54153519

about

Toxicity of chemical mixtures: proteomic analysis of persisting liver and kidney protein alterations induced by repeated exposure of rats to JP-8 jet fuel vapor.Methods for the characterization of Jet Propellent-8: vapor and aerosol.Biological and health effects of exposure to kerosene-based jet fuels and performance additives.DNA damage assessment by comet assay of human lymphocytes exposed to jet propulsion fuels.Identification of target genes responsive to JP-8 exposure in the rat central nervous system.In Vitro Pro-inflammatory Regulatory role of Substance P in Alveolar Macrophages and Type II Pneumocytes after JP-8 Exposure.Rat tissue and blood partition coefficients for n-alkanes (C8 to C12).Detection of oxidative species and low-molecular-weight DNA in skin following dermal exposure with JP-8 jet fuel.Age-related differences in pulmonary inflammatory responses to JP-8 jet fuel aerosol inhalation.Short-term pulmonary response to inhaled JP-8 jet fuel aerosol in mice.

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P2860

Neutral endopeptidase (NEP) and its role in pathological pulmonary change with inhalation exposure to JP-8 jet fuel.

http://www.wikidata.org/entity/Q54153519

description

1996 nî lūn-bûn

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A M Hays

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B J Tollinger

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H Chen

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J K Pfaff

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P2860

Neutral endopeptidase of a human airway epithelial cell line recovers after hypochlorous acid exposure: dexamethasone accelerates this by stimulating neutral endopeptidase mRNA synthesis

Toluene diisocyanate increases airway responsiveness to substance P and decreases airway neutral endopeptidase.

Cigarette smoke induces bronchoconstrictor hyperresponsiveness to substance P and inactivates airway neutral endopeptidase in the guinea pig. Possible role of free radicals

Neuropeptides in the respiratory tract. Part I.

Influenza infection causes airway hyperresponsiveness by decreasing enkephalinase.

Substance P primes human neutrophil activation: a mechanism for neurological regulation of inflammation.

Protective role of epithelium in the guinea pig airway.

Inhalation exposure to JP-8 jet fuel alters pulmonary function and substance P levels in Fischer 344 rats.

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