Stress-triggered abortion: inhibition of protective suppression and promotion of tumor necrosis factor-alpha (TNF-alpha) release as a mechanism triggering resorptions in mice.
about
To drive or be driven: the path of a mouse model of recurrent pregnancy lossTNF-alpha in pregnancy loss and embryo maldevelopment: a mediator of detrimental stimuli or a protector of the fetoplacental unit?Immunopathology of early pregnancyMaternal CD4⁺ and CD8⁺ T cell tolerance towards a fetal minor histocompatibility antigen in T cell receptor transgenic miceThe Adverse Effects of Auditory Stress on Mouse Uterus Receptivity and BehaviourCorticosterone response to gestational stress and postpartum memory function in mice.Demystifying animal models of adverse pregnancy outcomes: touching bench and bedside.Progesterone and HMOX-1 promote fetal growth by CD8+ T cell modulationEffect of Escherichia coli and Lactobacillus rhamnosus on macrophage inflammatory protein 3 alpha, tumor necrosis factor alpha, and transforming growth factor beta release by polarized rat uterine epithelial cells in culture.Adaptive responses of the maternal hypothalamic-pituitary-adrenal axis during pregnancy and lactation.Malaria-induced murine pregnancy failure: distinct roles for IFN-gamma and TNF.Stress and recurrent miscarriage.Maternal anti-placental cell-mediated reactivity and spontaneous abortions.Prenatal noise stress impairs HPA axis and cognitive performance in mice.Neuroendocrine circuitry and endometriosis: progesterone derivative dampens corticotropin-releasing hormone-induced inflammation by peritoneal cells in vitro.Inhibition of dipeptidyl peptidase IV (DP IV, CD26) activity abrogates stress-induced, cytokine-mediated murine abortions.Massage therapy effects on depressed pregnant women.The progesterone derivative dydrogesterone down-regulates neurokinin 1 receptor expression on lymphocytes, induces a Th2 skew and exerts hypoalgesic effects in mice.Nerve growth factor and its functional receptor TrkA are up-regulated in murine decidual tissue of stress-triggered and substance P-mediated abortion.Th1 cytokines and the prothrombinase fgl2 in stress-triggered and inflammatory abortion.Genetic factors in reproduction and their evolutionary significance.MD-1 is a critical part of the mechanism causing Th1-cytokine-triggered murine fetal loss syndrome.Stress triggered abortions are associated with alterations of granulated cells into the decidua.Tumor necrosis factor regulation of apoptosis in mouse preimplantation embryos and its antagonism by transforming growth factor alpha/phosphatidylionsitol 3-kinase signaling system.Stress and substance P but not the substance P-metabolite SP5-11 trigger murine abortion by augmenting TNF-alpha levels at the feto-maternal interface.Interleukin 10 regulates inflammatory cytokine synthesis to protect against lipopolysaccharide-induced abortion and fetal growth restriction in mice.
P2860
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P2860
Stress-triggered abortion: inhibition of protective suppression and promotion of tumor necrosis factor-alpha (TNF-alpha) release as a mechanism triggering resorptions in mice.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Stress-triggered abortion: inh ...... riggering resorptions in mice.
@en
Stress-triggered abortion: inh ...... of tumor necrosis factor-alpha
@nl
type
label
Stress-triggered abortion: inh ...... riggering resorptions in mice.
@en
Stress-triggered abortion: inh ...... of tumor necrosis factor-alpha
@nl
prefLabel
Stress-triggered abortion: inh ...... riggering resorptions in mice.
@en
Stress-triggered abortion: inh ...... of tumor necrosis factor-alpha
@nl
P2093
P2860
P1476
Stress-triggered abortion: inh ...... riggering resorptions in mice.
@en
P2093
P2860
P356
10.1111/J.1600-0897.1995.TB01141.X
P577
1995-01-01T00:00:00Z