Fumonisin- and AAL-Toxin-Induced Disruption of Sphingolipid Metabolism with Accumulation of Free Sphingoid Bases.
about
Ceramide synthases at the centre of sphingolipid metabolism and biologySecondary metabolites in fungus-plant interactionsNitric oxide-sphingolipid interplays in plant signalling: a new enigma from the Sphinx?The root hair assay facilitates the use of genetic and pharmacological tools in order to dissect multiple signalling pathways that lead to programmed cell deathThe conserved global regulator VeA is necessary for symptom production and mycotoxin synthesis in maize seedlings by Fusarium verticillioidesSphingolipids and plant defense/disease: the "death" connection and beyond.Arabidopsis ribosomal proteins control vacuole trafficking and developmental programs through the regulation of lipid metabolism.A post-genomic approach to understanding sphingolipid metabolism in Arabidopsis thalianaKnockout of Arabidopsis accelerated-cell-death11 encoding a sphingosine transfer protein causes activation of programmed cell death and defense.Long-chain bases, phosphatidic acid, MAPKs, and reactive oxygen species as nodal signal transducers in stress responses in Arabidopsis.Natural toxins for use in pest management.Nitric oxide production is not required for dihydrosphingosine-induced cell death in tobacco BY-2 cells.The involvement of jasmonates and ethylene in Alternaria alternata f. sp. lycopersici toxin-induced tomato cell death.Necrotroph attacks on plants: wanton destruction or covert extortion?Reactive oxygen species as transducers of sphinganine-mediated cell death pathway.A longevity assurance gene homolog of tomato mediates resistance to Alternaria alternata f. sp. lycopersici toxins and fumonisin B1.Ethylene Modulates Sphingolipid Synthesis in Arabidopsis.14-3-3-regulated Ca(2+)-dependent protein kinase CPK3 is required for sphingolipid-induced cell death in Arabidopsis.Human GLTP and mutant forms of ACD11 suppress cell death in the Arabidopsis acd11 mutant.Critical Role of COI1-Dependent Jasmonate Pathway in AAL toxin induced PCD in Tomato Revealed by Comparative ProteomicsThe role of the plasma membrane H+-ATPase in plant-microbe interactions.Modes of action of microbially-produced phytotoxins.Natural compounds as next-generation herbicides.Deciphering the link between salicylic acid signaling and sphingolipid metabolism.Alternaria Toxins: Potential Virulence Factors and Genes Related to Pathogenesis.Jaspine B induces nonapoptotic cell death in gastric cancer cells independently of its inhibition of ceramide synthase.Sphingolipids involvement in plant endomembrane differentiation: the BY2 case.Modifications of Sphingolipid Content Affect Tolerance to Hemibiotrophic and Necrotrophic Pathogens by Modulating Plant Defense Responses in Arabidopsis.Evidence for disruption of sphingolipid metabolism as a contributing factor in the toxicity and carcinogenicity of fumonisins.Unraveling the roles of sphingolipids in plant innate immunity.Involvement of sphingoid bases in mediating reactive oxygen intermediate production and programmed cell death in Arabidopsis.Subcellular compartmentalization of ceramide metabolism: MAM (mitochondria-associated membrane) and/or mitochondria?A general and concise asymmetric synthesis of sphingosine, safingol and phytosphingosines via tethered aminohydroxylation.Lesion mimic mutants: A classical, yet still fundamental approach to study programmed cell death.Susceptibility of Phelipanche and Orobanche species to AAL-toxin.Sphingolipids containing very-long-chain fatty acids define a secretory pathway for specific polar plasma membrane protein targeting in Arabidopsis.The plant disease resistance gene Asc-1 prevents disruption of sphingolipid metabolism during AAL-toxin-induced programmed cell death.Arabidopsis sphingosine kinase and the effects of phytosphingosine-1-phosphate on stomatal aperture.Apoptosis: A Functional Paradigm for Programmed Plant Cell Death Induced by a Host-Selective Phytotoxin and Invoked during Development.Survey of Alternaria toxin contamination in food from the German market, using a rapid HPLC-MS/MS approach.
P2860
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P2860
Fumonisin- and AAL-Toxin-Induced Disruption of Sphingolipid Metabolism with Accumulation of Free Sphingoid Bases.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@en
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@nl
type
label
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@en
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@nl
prefLabel
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@en
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@nl
P2093
P356
P1433
P1476
Fumonisin- and AAL-Toxin-Induc ...... ation of Free Sphingoid Bases.
@en
P2093
A. E. Sessions
A. H. Merrill
E. M. Wray
H. K. Abbas
J. K. Porter
R. T. Riley
S. O. Duke
P304
P356
10.1104/PP.106.3.1085
P407
P577
1994-11-01T00:00:00Z