Ultrastructural investigations of bone resorptive cells in two types of autosomal dominant osteopetrosis.
about
Characterization of osteoclasts from patients harboring a G215R mutation in ClC-7 causing autosomal dominant osteopetrosis type IIDifferentially expressed genes in autosomal dominant osteopetrosis type II osteoclasts reveal known and novel pathways for osteoclast biologyLevels of serotonin, sclerostin, bone turnover markers as well as bone density and microarchitecture in patients with high-bone-mass phenotype due to a mutation in Lrp5Dissociation of bone resorption and bone formation in adult mice with a non-functional V-ATPase in osteoclasts leads to increased bone strengthTenofovir-associated bone density loss.Resistin promotes the abnormal Type I collagen phenotype of subchondral bone in obese patients with end stage hip osteoarthritis.Osteoblast-specific Krm2 overexpression and Lrp5 deficiency have different effects on fracture healing in mice.Aging, human immunodeficiency virus, and bone health.The epithelial Ca2+ channel TRPV5 is essential for proper osteoclastic bone resorption.Acidification of the osteoclastic resorption compartment provides insight into the coupling of bone formation to bone resorption.Osteoclasts from patients with autosomal dominant osteopetrosis type I caused by a T253I mutation in low-density lipoprotein receptor-related protein 5 are normal in vitro, but have decreased resorption capacity in vivoLocalization of a gene for autosomal dominant osteopetrosis (Albers-Schönberg disease) to chromosome 1p21The bone resorption inhibitors odanacatib and alendronate affect post-osteoclastic events differently in ovariectomized rabbitsOsteoclast-derived coupling factors in bone remodeling.Novel targets for the prevention of osteoporosis - lessons learned from studies of metabolic bone disorders.Clcn7F318L/+ as a new mouse model of Albers-Schönberg disease.Locking plate osteosynthesis for a femoral fracture and subsequent nonunion in a patient with osteopetrosis
P2860
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P2860
Ultrastructural investigations of bone resorptive cells in two types of autosomal dominant osteopetrosis.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@en
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@nl
type
label
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@en
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@nl
prefLabel
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@en
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@nl
P2093
P1433
P1476
Ultrastructural investigations ...... osomal dominant osteopetrosis.
@en
P2093
J Bollerslev
L Mosekilde
S Pockwinse
P304
P356
10.1016/8756-3282(93)90316-3
P577
1993-11-01T00:00:00Z