Deciphering how HIV-1 weakens and cracks the bone.

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Deciphering how HIV-1 weakens and cracks the bone.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Deciphering how HIV-1 weakens and cracks the bone.

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Deciphering how HIV-1 weakens and cracks the bone.

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Deciphering how HIV-1 weakens and cracks the bone.

@en

Deciphering how HIV-1 weakens and cracks the bone.

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Deciphering how HIV-1 weakens and cracks the bone.

@en

Deciphering how HIV-1 weakens and cracks the bone.

@nl

P1476

Deciphering how HIV-1 weakens and cracks the bone.

@en

P2093

Ighovwerha Ofotokun

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P2860

Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

Bone resorption by osteoclasts

IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor kappa B ligand and tumor necrosis factor alpha from T cells

Narrowing the Gap in Life Expectancy Between HIV-Infected and HIV-Uninfected Individuals With Access to Care

Alterations in the immuno-skeletal interface drive bone destruction in HIV-1 transgenic rats.

Dysregulated B cell expression of RANKL and OPG correlates with loss of bone mineral density in HIV infection

B cells in HIV infection and disease

IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.

B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo

A novel T cell cytokine, secreted osteoclastogenic factor of activated T cells, induces osteoclast formation in a RANKL-independent manner.

P304

2551-2553

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P31

scholarly article

P356

10.1073/PNAS.1801555115

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P407

English

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P478

115

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P577

2018-03-01T00:00:00Z

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29496959

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P932

5856569

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