Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice. - Wikidata - wikimedia - TriplyDB

Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice.

Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice.

http://www.wikidata.org/entity/Q54292231

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Kinetic mRNA Profiling in a Rat Model of Left-Ventricular Hypertrophy Reveals Early Expression of Chemokines and Their Receptors Toll-Like Receptor 9 Promotes Survival in SERCA2a KO Heart Failure Mice.Toll-like receptor 9 mediated responses in cardiac fibroblasts Early life exposure to air pollution induces adult cardiac dysfunction.Ly6C(low) and not Ly6C(high) macrophages accumulate first in the heart in a model of murine pressure-overload.Sustained Toll-Like Receptor 9 Activation Promotes Systemic and Cardiac Inflammation, and Aggravates Diastolic Heart Failure in SERCA2a KO Mice Proinflammatory Stimulation of Toll-Like Receptor 9 with High Dose CpG ODN 1826 Impairs Endothelial Regeneration and Promotes Atherosclerosis in Mice.Extracellular mtDNA activates NF-κB via toll-like receptor 9 and induces cell death in cardiomyocytes.Tlr2 deficiency does not limit the development of left ventricular hypertrophy in a model of transverse aortic constriction induced pressure overload In vivo TLR9 inhibition attenuates CpG-induced myocardial dysfunction.Cellular and molecular studies of the effects of a selective COX-2 inhibitor celecoxib in the cardiac cell line H9c2 and their correlation with death mechanisms.Myocyte signalling in leucocyte recruitment to the heart.Emerging roles of innate immune signaling and toll-like receptors in fibrosis and systemic sclerosis.Cardiac Hypertrophy: An Introduction to Molecular and Cellular Basis.Simultaneous adrenal and cardiac g-protein-coupled receptor-gβγ inhibition halts heart failure progression.Cellular interplay via cytokine hierarchy causes pathological cardiac hypertrophy in RAF1-mutant Noonan syndrome.Pre-conditioning with synthetic CpG-oligonucleotides attenuates myocardial ischemia/reperfusion injury via IL-10 up-regulation.Metallothioneins 1 and 2 Modulate Inflammation and Support Remodeling in Ischemic Cardiomyopathy in Mice.Pirfenidone attenuates cardiac fibrosis in a mouse model of TAC-induced left ventricular remodeling by suppressing NLRP3 inflammasome formation.CpG oligodeoxynucleotide preconditioning improves cardiac function after myocardial infarction via modulation of energy metabolism and angiogenesis.The Role of Toll-Like Receptor Signaling in the Progression of Heart Failure.Overexpression of Cx43 in cells of the myocardial scar: Correction of post-infarct arrhythmias through heterotypic cell-cell coupling.Inflammatory Mediators Drive Adverse Right Ventricular Remodeling and Dysfunction and Serve as Potential Biomarkers.Synthetic CpG oligonucleotides induce a genetic profile ameliorating murine myocardial I/R injury.

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P2860

Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice.

http://www.wikidata.org/entity/Q54292231

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年学术文章

@wuu

2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@en

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@nl

type

label

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@en

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@nl

prefLabel

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@en

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@nl

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Cardiovascular Research

P1476

Priming with synthetic oligonu ...... d cardiac hypertrophy in mice.

@en

P2093

Andreas Hoeft

http://www.w3.org/2001/XMLSchema#string

Georg Baumgarten

http://www.w3.org/2001/XMLSchema#string

Georg D Duerr

http://www.w3.org/2001/XMLSchema#string

Jan Mersmann

http://www.w3.org/2001/XMLSchema#string

Julia Schild

http://www.w3.org/2001/XMLSchema#string

Kai Zacharowski

http://www.w3.org/2001/XMLSchema#string

Markus Velten

http://www.w3.org/2001/XMLSchema#string

Olaf Boehm

http://www.w3.org/2001/XMLSchema#string

Oliver Dewald

http://www.w3.org/2001/XMLSchema#string

Pascal Knuefermann

http://www.w3.org/2001/XMLSchema#string

P2860

Normalization for cDNA microarray data: a robust composite method addressing single and multiple slide systematic variation.

Dynamic molecular processes mediate cellular mechanotransduction.

Load-dependent and -independent regulation of proinflammatory cytokine and cytokine receptor gene expression in the adult mammalian heart.

Toll-like receptor 4 modulates myocardial ischaemia-reperfusion injury: Role of matrix metalloproteinases.

Short- and long-term changes in gene expression mediated by the activation of TLR9

Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium.

Induction of cardiac fibrosis by transforming growth factor-beta(1).

Myocardin-related transcription factor-a controls myofibroblast activation and fibrosis in response to myocardial infarction.

CpG motifs in bacterial DNA trigger direct B-cell activation.

CD14 is a coreceptor of Toll-like receptors 7 and 9.

P304

422-432

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scholarly article

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10.1093/CVR/CVS280

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3

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96

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2012-09-12T00:00:00Z

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230850362

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22977006

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