Defects in insulin binding and receptor kinase in cells from a woman with type A insulin resistance and from her family.

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Defects in insulin binding and receptor kinase in cells from a woman with type A insulin resistance and from her family.

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http://www.wikidata.org/entity/Q54303457

description

1991 nî lūn-bûn

@nan

1991年の論文

@ja

1991年学术文章

@wuu

1991年学术文章

@zh-cn

1991年学术文章

@zh-hans

1991年学术文章

@zh-my

1991年学术文章

@zh-sg

1991年學術文章

@yue

1991年學術文章

@zh

1991年學術文章

@zh-hant

name

Defects in insulin binding and ...... esistance and from her family.

@en

Defects in insulin binding and ...... esistance and from her family.

@nl

type

Item

label

Defects in insulin binding and ...... esistance and from her family.

@en

Defects in insulin binding and ...... esistance and from her family.

@nl

prefLabel

Defects in insulin binding and ...... esistance and from her family.

@en

Defects in insulin binding and ...... esistance and from her family.

@nl

P1476

Defects in insulin binding and ...... esistance and from her family.

@en

P2093

F Shimada

http://www.w3.org/2001/XMLSchema#string

H Makino

http://www.w3.org/2001/XMLSchema#string

M Mimura

http://www.w3.org/2001/XMLSchema#string

M Taira

http://www.w3.org/2001/XMLSchema#string

M Tawata

http://www.w3.org/2001/XMLSchema#string

N Hashimoto

http://www.w3.org/2001/XMLSchema#string

O Nozaki

http://www.w3.org/2001/XMLSchema#string

S Yoshida

http://www.w3.org/2001/XMLSchema#string

T Onaya

http://www.w3.org/2001/XMLSchema#string

Y Suzuki

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P2860

Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucose

The human insulin receptor cDNA: the structural basis for hormone-activated transmembrane signalling

Human insulin receptor and its relationship to the tyrosine kinase family of oncogenes

Properties of the insulin receptor ectodomain

Detection of an alteration in the insulin-receptor gene in a patient with insulin resistance, acanthosis nigricans, and the polycystic ovary syndrome (type A insulin resistance).

Acute insulin action requires insulin receptor kinase activity: introduction of an inhibitory monoclonal antibody into mammalian cells blocks the rapid effects of insulin.

Replacement of lysine residue 1030 in the putative ATP-binding region of the insulin receptor abolishes insulin- and antibody-stimulated glucose uptake and receptor kinase activity.

The syndromes of insulin resistance and acanthosis nigricans. Insulin-receptor disorders in man.

Individuals with only one allele for a functional insulin receptor have a tendency to hyperinsulinaemia but not to hyperglycaemia.

Characterization of binding and phosphorylation defects of erythrocyte insulin receptors in the type A syndrome of insulin resistance.

P2888

bf00500378

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86-92

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scholarly article

P356

10.1007/BF00500378

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P478

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P577

1991-02-01T00:00:00Z

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P698

1648521

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