Rosmarinic acid exerts an antiosteoporotic effect in the RANKL-induced mouse model of bone loss by promotion of osteoblastic differentiation and inhibition of osteoclastic differentiation.

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Solanum Muricatum Promotes Osteogenic Differentiation of Rat Bone Marrow Stromal Cells.

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Q48286221-3769C6C9-A098-4E05-A704-2BCB4D7FEBC5

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Rosmarinic acid exerts an antiosteoporotic effect in the RANKL-induced mouse model of bone loss by promotion of osteoblastic differentiation and inhibition of osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... osteoclastic differentiation.

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Rosmarinic acid exerts an anti ...... f osteoclastic differentiation

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Akira Yamaguchi

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Byung-Yoon Cha

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Je-Tae Woo

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Ji-Won Lee

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Midori Asai

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Sang-Kyung Jeon

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Takayuki Yonezawa

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P2860

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Bone morphogenetic proteins

The spice sage and its active ingredient rosmarinic acid protect PC12 cells from amyloid-beta peptide-induced neurotoxicity

Osteoclast differentiation and activation

Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts

Orally administered rosmarinic acid is present as the conjugated and/or methylated forms in plasma, and is degraded and metabolized to conjugated forms of caffeic acid, ferulic acid and m-coumaric acid.

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386-400

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scholarly article

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10.1002/MNFR.201400164

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Tadahiro Iimura

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2015-01-21T00:00:00Z

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25380345

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Rosmarinic acid exerts an antiosteoporotic effect in the RANKL-induced mouse model of bone loss by promotion of osteoblastic differentiation and inhibition of osteoclastic differentiation.

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P2860

P2860

Rosmarinic acid exerts an antiosteoporotic effect in the RANKL-induced mouse model of bone loss by promotion of osteoblastic differentiation and inhibition of osteoclastic differentiation.

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