β3 adrenergic receptor selective stimulation during ischemia/reperfusion improves cardiac function in translational models through inhibition of mPTP opening in cardiomyocytes.
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Imbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in miceNeutrophil stunning by metoprolol reduces infarct sizeChronic β1-adrenergic blockade enhances myocardial β3-adrenergic coupling with nitric oxide-cGMP signaling in a canine model of chronic volume overload: new insight into mechanisms of cardiac benefit with selective β1-blocker therapy.Beta-3 adrenergic agonists reduce pulmonary vascular resistance and improve right ventricular performance in a porcine model of chronic pulmonary hypertension.Exogenous Administration of Recombinant MIF at Physiological Concentrations Failed to Attenuate Infarct Size in a Langendorff Perfused Isolated Mouse Heart ModelCardiac salvage by tweaking with beta-3-adrenergic receptors.Targeting β3-Adrenergic Receptors in the Heart: Selective Agonism and β-Blockade.New and Emerging Therapies and Targets: Beta-3 Agonists.A Novel Systems-Biology Algorithm for the Analysis of Coordinated Protein Responses Using Quantitative ProteomicsIntracoronary Administration of Allogeneic Adipose Tissue-Derived Mesenchymal Stem Cells Improves Myocardial Perfusion But Not Left Ventricle Function, in a Translational Model of Acute Myocardial Infarction.Protective approaches against myocardial ischemia reperfusion injuryOptimization of dual-saturation single bolus acquisition for quantitative cardiac perfusion and myocardial blood flow maps.Rosa rugosa flavonoids alleviate myocardial ischemia reperfusion injury in mice by suppressing JNK and p38 MAPK.Bloodless reperfusion with the oxygen carrier HBOC-201 in acute myocardial infarction: a novel platform for cardioprotective probes delivery.Therapeutic inhibition of miR-375 attenuates post-myocardial infarction inflammatory response and left ventricular dysfunction via PDK-1-AKT signalling axis.β1-Blockade Prevents Post-Ischemic Myocardial Decompensation Via β3AR-Dependent Protective Sphingosine-1 Phosphate Signaling.Exercise does not activate the β3 adrenergic receptor-eNOS pathway, but reduces inducible NOS expression to protect the heart of obese diabetic mice.
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P2860
β3 adrenergic receptor selective stimulation during ischemia/reperfusion improves cardiac function in translational models through inhibition of mPTP opening in cardiomyocytes.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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2014年學術文章
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2014年學術文章
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name
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@en
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@nl
type
label
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@en
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@nl
prefLabel
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@en
β3 adrenergic receptor selecti ...... PTP opening in cardiomyocytes.
@nl
P2093
P2860
P50
P1476
β3 adrenergic receptor selecti ...... mPTP opening in cardiomyocytes
@en
P2093
Ana García-Alvarez
Andrés Pun
Carlos Macaya
Jaime García-Prieto
Jose Manuel García-Ruiz
Mario Nuno-Ayala
P2860
P2888
P356
10.1007/S00395-014-0422-0
P50
P577
2014-06-21T00:00:00Z