MAPK signaling is required for LPS-induced VEGF in pulp stem cells. - Wikidata - wikimedia - TriplyDB

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

http://www.wikidata.org/entity/Q54391382

about

Genome-wide association scan of dental caries in the permanent dentition.C-reactive protein can upregulate VEGF expression to promote ADSC-induced angiogenesis by activating HIF-1α via CD64/PI3k/Akt and MAPK/ERK signaling pathways.The herpes simplex virus-1 transactivator infected cell protein-4 drives VEGF-A dependent neovascularization.Genomic meta-analysis of growth factor and integrin pathways in chronic kidney transplant injury Melatonin attenuates inflammation of acute pulpitis subjected to dental pulp injury.An Overview of Pathogen Recognition Receptors for Innate Immunity in Dental Pulp.Effects of human vascular endothelial growth factor on reparative dentin formation.Regenerative endodontics in light of the stem cell paradigm.Dentin-pulp complex regeneration: from lab to clinic.Regenerative endodontics.Attenuation of LPS-induced cyclooxygenase-2 and inducible NO synthase expression by lysophosphatidic acid in macrophages.Evaluation of the expression of VIII factor and VEGF in the regeneration of non-vital teeth in dogs using propolis.Dental follicle progenitor cells responses to Porphyromonas gingivalis LPS.Mechanical stretch increases the proliferation while inhibiting the osteogenic differentiation in dental pulp stem cells.Activation of TLR4 induces VEGF expression via Akt pathway in nasal polyps.LPS promote the odontoblastic differentiation of human dental pulp stem cells via MAPK signaling pathway.TNF-α and LPS activate angiogenesis via VEGF and SIRT1 signalling in human dental pulp cells.Repeated lipopolysaccharide stimulation promotes cellular senescence in human dental pulp stem cells (DPSCs).Simvastatin and nanofibrous poly(l-lactic acid) scaffolds to promote the odontogenic potential of dental pulp cells in an inflammatory environment.LPS induces IL-8 expression through TLR4, MyD88, NF-kappaB and MAPK pathways in human dental pulp stem cells.Repeated stimulation by LPS promotes the senescence of DPSCs via TLR4/MyD88-NF-κB-p53/p21 signaling.Static magnetic field attenuates lipopolysaccharide-induced inflammation in pulp cells by affecting cell membrane stability.Sphingosine-1-phosphate mediates AKT/ERK maintenance of dental pulp homoeostasis.Vascular endothelial growth factors signalling in normal human dental pulp: a study of gene and protein expression.

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P2860

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

http://www.wikidata.org/entity/Q54391382

description

2010 nî lūn-bûn

@nan

2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh

2010年學術文章

@zh-hant

name

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@en

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@nl

type

label

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@en

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@nl

prefLabel

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@en

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@nl

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0022034509357556

P1433

Journal of Dental Research

P1476

MAPK signaling is required for LPS-induced VEGF in pulp stem cells.

@en

P2093

C E Shelburne

http://www.w3.org/2001/XMLSchema#string

D Vodopyanov

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J E Nör

http://www.w3.org/2001/XMLSchema#string

J S Son

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M Hasegawa

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T M Botero

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P2860

The structural basis of lipopolysaccharide recognition by the TLR4-MD-2 complex

Vascular endothelial growth factor is a secreted angiogenic mitogen

Postnatal human dental pulp stem cells (DPSCs) in vitro and in vivo

Quantitative microbiological study of human carious dentine by culture and real-time PCR: association of anaerobes with histopathological changes in chronic pulpitis.

Structural and functional analyses of bacterial lipopolysaccharides.

NF-kappaB activation in human dental pulp stem cells by TNF and LPS.

Vascular endothelial growth factor (VEGF)-mediated angiogenesis is associated with enhanced endothelial cell survival and induction of Bcl-2 expression

TLR4 mediates LPS-induced VEGF expression in odontoblasts.

Microbiologic factors in endodontology.

Effect of lipopolysaccharides on vascular endothelial growth factor expression in mouse pulp cells and macrophages.

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264-269

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10.1177/0022034509357556

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3

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20110511

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