HMGCoA reductase inhibition reverses myocardial fibrosis and diastolic dysfunction through AMP-activated protein kinase activation in a mouse model of metabolic syndrome.
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Statin-Induced Increases in Atrophy Gene Expression Occur Independently of Changes in PGC1α Protein and Mitochondrial ContentCardiac fibrosis in myocardial infarction-from repair and remodeling to regenerationQiliqiangxin Enhances Cardiac Glucose Metabolism and Improves Diastolic Function in Spontaneously Hypertensive RatsA cell-based screening assay to identify pharmaceutical compounds that enhance the regenerative quality of corneal repair.Simvastatin alleviates cardiac fibrosis induced by infarction via up-regulation of TGF-β receptor III expression.Cardiomyocyte changes in the metabolic syndrome and implications for endogeneous protective strategies.Activation of AMP-activated protein kinase inhibits ER stress and renal fibrosis.AMPK in Cardiovascular Diseases.CTRP3 attenuates post-infarct cardiac fibrosis by targeting Smad3 activation and inhibiting myofibroblast differentiation.Hypertension is a conditional factor for the development of cardiac hypertrophy in type 2 diabetic mice.AMPK: a novel target for treating hepatic fibrosisAMPK and cardiac remodelling.Effectiveness of Cardiac Rehabilitation for Prevention and Treatment of Sarcopenia in Patients with Cardiovascular Disease - A Retrospective Cross-Sectional Analysis.Endothelial Nitric Oxide Synthase-Independent Pleiotropic Effects of Pitavastatin Against Atherogenesis and Limb Ischemia in Mice.Rosmarinic acid attenuates cardiac fibrosis following long-term pressure overload via AMPKα/Smad3 signaling.PTEN upregulation may explain the development of insulin resistance and type 2 diabetes with high dose statins.Molecular and cellular mechanisms of glucagon-like peptide-1 receptor agonist-mediated attenuation of cardiac fibrosis.Effects of 12 weeks of atorvastatin therapy on myocardial fibrosis and circulating fibrosis biomarkers in statin-naïve patients with hypertension with atherosclerosis.Asymptomatic Left Ventricular Dysfunction and Metabolic Syndrome: Results from an Italian Multicenter Study
P2860
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P2860
HMGCoA reductase inhibition reverses myocardial fibrosis and diastolic dysfunction through AMP-activated protein kinase activation in a mouse model of metabolic syndrome.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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name
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@en
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@nl
type
label
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@en
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@nl
prefLabel
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@en
HMGCoA reductase inhibition re ...... e model of metabolic syndrome.
@nl
P2093
P2860
P356
P1476
HMGCoA reductase inhibition re ...... se model of metabolic syndrome
@en
P2093
Andreas Markl
Annelies Vanderper
Christophe Beauloye
Denise Hilfiker-Kleiner
Gauthier Noppe
Julien Hamelet
Marc van Bilsen
Nerea Hermida
Sandrine Horman
Tim Van Assche
P2860
P356
10.1093/CVR/CVT070
P577
2013-03-29T00:00:00Z