Epithelial-mesenchymal transition as a potential explanation for podocyte depletion in diabetic nephropathy. - Wikidata - wikimedia - TriplyDB

Epithelial-mesenchymal transition as a potential explanation for podocyte depletion in diabetic nephropathy.

Epithelial-mesenchymal transition as a potential explanation for podocyte depletion in diabetic nephropathy.

http://www.wikidata.org/entity/Q54472919

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Growth hormone (GH)-dependent expression of a natural antisense transcript induces zinc finger E-box-binding homeobox 2 (ZEB2) in the glomerular podocyte: a novel action of gh with implications for the pathogenesis of diabetic nephropathy A Systems Biology Overview on Human Diabetic Nephropathy: From Genetic Susceptibility to Post-Transcriptional and Post-Translational Modifications Epithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?Dedifferentiation of immortalized human podocytes in response to transforming growth factor-β: a model for diabetic podocytopathy.Urinary podocyte-associated mRNA profile in various stages of diabetic nephropathy.TGF-β-activated kinase 1 is crucial in podocyte differentiation and glomerular capillary formation.Chronic kidney disease prediction is an inexact science: The concept of "progressors" and "nonprogressors"Blockade of Wnt/β-catenin signaling by paricalcitol ameliorates proteinuria and kidney injury.Inhibition of integrin-linked kinase blocks podocyte epithelial-mesenchymal transition and ameliorates proteinuria.Hepatocyte growth factor signaling ameliorates podocyte injury and proteinuria.Epithelial to mesenchymal transition as a biomarker in renal fibrosis: are we ready for the bedside?Canonical Wnt/β-catenin signaling mediates transforming growth factor-β1-driven podocyte injury and proteinuria.A plasminogen activator inhibitor type 1 mutant retards diabetic nephropathy in db/db mice by protecting podocytes Urinary FSP1 is a biomarker of crescentic GN Implication of CD38 gene in podocyte epithelial-to-mesenchymal transition and glomerular sclerosis.Genomic biomarkers for chronic kidney disease.Cellular and molecular mechanisms of renal fibrosis.New insights into epithelial-mesenchymal transition in kidney fibrosis.Sirolimus modulates HIVAN phenotype through inhibition of epithelial mesenchymal transition.Tangzhiqing Granules Alleviate Podocyte Epithelial-Mesenchymal Transition in Kidney of Diabetic Rats Wnt/β-catenin signalling and podocyte dysfunction in proteinuric kidney disease.Transforming growth factor-beta and the glomerular filtration barrier.Advanced glycation end-products reduce podocyte adhesion by activating the renin-angiotensin system and increasing integrin-linked kinase.The coordinated roles of miR-26a and miR-30c in regulating TGFβ1-induced epithelial-to-mesenchymal transition in diabetic nephropathy.A glimpse of the pathogenetic mechanisms of Wnt/β-catenin signaling in diabetic nephropathy.The role of metabolic and haemodynamic factors in podocyte injury in diabetes.WT1 in disease: shifting the epithelial-mesenchymal balance.Relationship between urinary podocytes and kidney diseases.Renoprotection and the Bardoxolone Methyl Story - Is This the Right Way Forward? A Novel View of Renoprotection in CKD Trials: A New Classification Scheme for Renoprotective Agents.Unlocking the biology of RAGE in diabetic microvascular complications Signal transduction in podocytes--spotlight on receptor tyrosine kinases.Dipping your feet in the water: podocytes in urine.Molecular and cellular events mediating glomerular podocyte dysfunction and depletion in diabetes mellitus.Podocyte dedifferentiation: a specialized process for a specialized cell.Renin-angiotensin system within the diabetic podocyte.Diverse origins of the myofibroblast—implications for kidney fibrosis.Exosomes from high glucose-treated glomerular endothelial cells trigger the epithelial-mesenchymal transition and dysfunction of podocytes CTGF mediates high-glucose induced epithelial-mesenchymal transition through activation of β-catenin in podocytes.Critical role of serum response factor in podocyte epithelial-mesenchymal transition of diabetic nephropathy.The roles of connective tissue growth factor and integrin-linked kinase in high glucose-induced phenotypic alterations of podocytes.

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P2860

Epithelial-mesenchymal transition as a potential explanation for podocyte depletion in diabetic nephropathy.

http://www.wikidata.org/entity/Q54472919

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

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Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

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type

label

Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

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Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

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prefLabel

Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

@en

Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

@nl

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J.AJKD.2009.05.009

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American Journal of Kidney Diseases

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Epithelial-mesenchymal transit ...... etion in diabetic nephropathy.

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Atsushi Kubo

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Daisuke Suzuki

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Eric G Neilson

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Kimihiko Nakatani

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Koji Harada

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Kuniko Kimura

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Masao Kanauchi

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Masao Toyoda

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Masayuki Iwano

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Yasuhiro Akai

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653-664

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scholarly article

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10.1053/J.AJKD.2009.05.009

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4

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19615802

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