Prevention of neural tube defects in Lrp2 mutant mouse embryos by folic acid supplementation.

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Insights into the Etiology of Mammalian Neural Tube Closure Defects from Developmental, Genetic and Evolutionary Studies

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Prevention of neural tube defects in Lrp2 mutant mouse embryos by folic acid supplementation.

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2017 nî lūn-bûn

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Prevention of neural tube defe ...... by folic acid supplementation.

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Prevention of neural tube defe ...... by folic acid supplementation.

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Prevention of neural tube defe ...... by folic acid supplementation.

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Prevention of neural tube defe ...... by folic acid supplementation.

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Bethany A Stokes

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Irene E Zohn

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Julia A Sabatino

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Updated national birth prevalence estimates for selected birth defects in the United States, 2004-2006

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Megalin-dependent cubilin-mediated endocytosis is a major pathway for the apical uptake of transferrin in polarized epithelia

A review of Donnai-Barrow and facio-oculo-acoustico-renal (DB/FOAR) syndrome: clinical features and differential diagnosis

Neural tube defects and folate: case far from closed

The iron exporter ferroportin 1 is essential for development of the mouse embryo, forebrain patterning and neural tube closure

Defective forebrain development in mice lacking gp330/megalin.

The flatiron mutation in mouse ferroportin acts as a dominant negative to cause ferroportin disease

Reduced-folate carrier (RFC) is expressed in placenta and yolk sac, as well as in cells of the developing forebrain, hindbrain, neural tube, craniofacial region, eye, limb buds and heart

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scholarly article

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10.1002/BDRA.23589

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109

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Prevention of neural tube defects in Lrp2 mutant mouse embryos by folic acid supplementation.

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P2860

P2860

Prevention of neural tube defects in Lrp2 mutant mouse embryos by folic acid supplementation.

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