BRAFV600E mutation, TIMP-1 upregulation, and NF-κB activation: closing the loop on the papillary thyroid cancer trilogy.
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Signaling Pathways in Thyroid Cancer and Their Therapeutic ImplicationsSignaling and stress: The redox landscape in NOS2 biologyBRAF mutation in papillary thyroid carcinomaRAC1b overexpression stimulates proliferation and NF-kB-mediated anti-apoptotic signaling in thyroid cancer cells.LDOC1 inhibits proliferation and promotes apoptosis by repressing NF-κB activation in papillary thyroid carcinomaClinical utility of measuring expression levels of KAP1, TIMP1 and STC2 in peripheral blood of patients with gastric cancerSerum biomarkers of papillary thyroid cancerRNA sequencing identifies multiple fusion transcripts, differentially expressed genes, and reduced expression of immune function genes in BRAF (V600E) mutant vs BRAF wild-type papillary thyroid carcinoma.Cardiac fibrosis in mouse expressing DsRed tetramers involves chronic autophagy and proteasome degradation insufficiency.Translational research in endocrine surgery.The association between BRAF (V600E) mutation and pathological features in PTC.Anaplastic Thyroid Carcinoma: A ceRNA Analysis Pointed to a Crosstalk between SOX2, TP53, and microRNA Biogenesis.MicroRNAs in the thyroid.Claudin-7 suppresses the cytotoxicity of TRAIL-expressing mesenchymal stem cells in H460 human non-small cell lung cancer cells.In papillary thyroid carcinoma, TIMP-1 expression correlates with BRAF (V600E) mutation status and together with hypoxia-related proteins predicts aggressive behavior.Therapeutic potential of Wenshen Xiaozheng tang, a traditional Chinese medicine prescription, for treating endometriosis.BRAFV600E and RET/PTC Promote Proliferation and Migration of Papillary Thyroid Carcinoma Cells In Vitro by Regulating Nuclear Factor-κB.Whole-genome profiling highlights the molecular complexity underlying eccentric cardiac hypertrophy.Sodium iodide symporter in the fight against thyroid cancer.The Major Role of NF-kB in the Depth of Invasion on Acral Melanoma by Decreasing CD8+ T Cells.Overexpression of miR-221 is associated with aggressive clinicopathologic characteristics and the BRAF mutation in papillary thyroid carcinomas.BRAF V600E and RET/PTC Promote the Activity of Nuclear Factor-κB, Inflammatory Mediators, and Lymph Node Metastasis in Papillary Thyroid Carcinoma: A Study of 50 Patients in Inner Mongolia
P2860
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P2860
BRAFV600E mutation, TIMP-1 upregulation, and NF-κB activation: closing the loop on the papillary thyroid cancer trilogy.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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name
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@en
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@nl
type
label
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@en
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@nl
prefLabel
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@en
BRAFV600E mutation, TIMP-1 upr ...... illary thyroid cancer trilogy.
@nl
P2093
P50
P356
P1476
BRAFV600E mutation, TIMP-1 upr ...... pillary thyroid cancer trilogy
@en
P2093
Aldo Galluzzo
Alessandra Bommarito
Angela Criscimanna
Elvira Carissimi
Giovanni Zito
Giuseppe Modica
M Adelfio Latteri
Marco C Amato
Maria Pitrone
Monica Zerilli
P304
P356
10.1530/ERC-11-0076
P577
2011-11-14T00:00:00Z