Deleterious Effect of Advanced CKD on Glyoxalase System Activity not Limited to Diabetes Aetiology.

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Deleterious Effect of Advanced CKD on Glyoxalase System Activity not Limited to Diabetes Aetiology.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

@en

Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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P1476

Deleterious Effect of Advanced ...... Limited to Diabetes Aetiology.

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P2093

Jitka Řehořová

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Katarína Chalásová

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Lukáš Pácal

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P2860

Dicarbonyl stress in cell and tissue dysfunction contributing to ageing and disease

The formation of methylglyoxal from triose phosphates. Investigation using a specific assay for methylglyoxal.

Overexpression of glyoxalase-I reduces hyperglycemia-induced levels of advanced glycation end products and oxidative stress in diabetic rats

Knockdown of glyoxalase 1 mimics diabetic nephropathy in nondiabetic mice.

Methylglyoxal-Glyoxalase 1 Balance: The Root of Vascular Damage.

Methylglyoxal, glyoxalase 1 and the dicarbonyl proteome.

Glycative stress and glyoxalase in kidney disease and aging.

Epidemiology and risk factors for diabetic kidney disease.

Increased glyoxalase I levels inhibit accumulation of oxidative stress and an advanced glycation end product in mouse mesangial cells cultured in high glucose.

Glyoxalase I overexpression ameliorates renal ischemia-reperfusion injury in rats.

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scholarly article

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10.3390/IJMS19051517

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English

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P478

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P50

Josef Tomandl

Kateřina Kaňková

Anna Pleskacova

P577

2018-05-18T00:00:00Z

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P698

29783710

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P932

5983829

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