NOTCH3 inactivation increases triple negative breast cancer sensitivity to gefitinib by promoting EGFR tyrosine dephosphorylation and its intracellular arrest.

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NOTCH3 inactivation increases triple negative breast cancer sensitivity to gefitinib by promoting EGFR tyrosine dephosphorylation and its intracellular arrest.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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NOTCH3 inactivation increases ...... and its intracellular arrest.

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NOTCH3 inactivation increases ...... and its intracellular arrest.

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NOTCH3 inactivation increases ...... and its intracellular arrest.

@en

NOTCH3 inactivation increases ...... and its intracellular arrest.

@nl

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NOTCH3 inactivation increases ...... and its intracellular arrest.

@en

NOTCH3 inactivation increases ...... and its intracellular arrest.

@nl

P1476

NOTCH3 inactivation increases ...... and its intracellular arrest.

@en

P2093

Alessandra Vacca

http://www.w3.org/2001/XMLSchema#string

Carlo Capalbo

http://www.w3.org/2001/XMLSchema#string

Claudio Talora

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Diana Bellavia

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Eugenia Giuliani

http://www.w3.org/2001/XMLSchema#string

Giulia Diluvio

http://www.w3.org/2001/XMLSchema#string

Giulia d'Amati

http://www.w3.org/2001/XMLSchema#string

Marella Maroder

http://www.w3.org/2001/XMLSchema#string

Maria Gemma Pignataro

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Maria Valeria Giuli

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P2860

Receptor-type protein-tyrosine phosphatase-kappa regulates epidermal growth factor receptor function

Notch and EGFR pathway interaction regulates neural stem cell number and self-renewal

PTPH1 dephosphorylates and cooperates with p38gamma MAPK to increase ras oncogenesis through PDZ-mediated interaction

Phosphorylated EGFR expression may predict outcome of EGFR-TKIs therapy for the advanced NSCLC patients with wild-type EGFR

Analysis of tumor specimens at the time of acquired resistance to EGFR-TKI therapy in 155 patients with EGFR-mutant lung cancers.

A perspective on anti-EGFR therapies targeting triple-negative breast cancer

High-level coexpression of JAG1 and NOTCH1 is observed in human breast cancer and is associated with poor overall survival

Phosphotyrosine 1173 mediates binding of the protein-tyrosine phosphatase SHP-1 to the epidermal growth factor receptor and attenuation of receptor signaling

Src family kinases mediate epidermal growth factor receptor signaling from lipid rafts in breast cancer cells.

Lipid raft localization of EGFR alters the response of cancer cells to the EGFR tyrosine kinase inhibitor gefitinib

P2888

s41389-018-0051-9

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scholarly article

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10.1038/S41389-018-0051-9

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P478

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P50

Zein Mersini Besharat

Giulia Franciosa

Francesca Del Gaudio

Saula Checquolo

P577

2018-05-25T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1104163215

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29795369

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5968025

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