COX-2 is required for the modulation of spinal nociceptive information related to ephrinB/EphB signalling.

about

COX-2 is required for the modulation of spinal nociceptive information related to ephrinB/EphB signalling.

Item

http://www.wikidata.org/entity/Q55060270

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年学术文章

@wuu

2015年学术文章

@zh-cn

2015年学术文章

@zh-hans

2015年学术文章

@zh-my

2015年学术文章

@zh-sg

2015年學術文章

@yue

2015年學術文章

@zh

2015年學術文章

@zh-hant

name

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@en

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@nl

type

Item

label

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@en

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@nl

prefLabel

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@en

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@nl

P1476

COX-2 is required for the modu ...... ed to ephrinB/EphB signalling.

@en

P2093

C-J Zhang

http://www.w3.org/2001/XMLSchema#string

J-L Cao

http://www.w3.org/2001/XMLSchema#string

L-N Yu

http://www.w3.org/2001/XMLSchema#string

M Yan

http://www.w3.org/2001/XMLSchema#string

X-L Zhou

http://www.w3.org/2001/XMLSchema#string

Y Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Sensitization of TRPV1 by EP1 and IP reveals peripheral nociceptive mechanism of prostaglandins.

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Mechanisms and functions of Eph and ephrin signalling

Eph receptors in the adult brain

Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice

Quantitative assessment of tactile allodynia in the rat paw

Eph-ephrin bidirectional signaling in physiology and disease

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

Interleukin-1beta-mediated induction of Cox-2 in the CNS contributes to inflammatory pain hypersensitivity.

Nuclear calcium signaling in spinal neurons drives a genomic program required for persistent inflammatory pain

P304

1277-1287

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/EJP.657

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-04-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

25919495

http://www.w3.org/2001/XMLSchema#string