Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
about
T3 inhibits the calcification of vascular smooth muscle cells and the potential mechanism.SM22α inhibits lamellipodium formation and migration via Ras-Arp2/3 signaling in synthetic VSMCs.Insulin-independent GLUT4 translocation in proliferative vascular smooth muscle cells involves SM22α.Short-Term Angiotensin II Treatment Affects Large Artery Biomechanics and Function in the Absence of Small Artery Alterations in Mice.Immunoproteasome Inhibition Impairs T and B Cell Activation by Restraining ERK Signaling and Proteostasis
P2860
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
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2014年学术文章
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2014年學術文章
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2014年學術文章
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name
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@en
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@nl
type
label
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@en
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@nl
prefLabel
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@en
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@nl
P2093
P2860
P1476
Smooth muscle 22α facilitates angiotensin II-induced signaling and vascular contraction.
@en
P2093
Li-Li Zhao
Sui-Bing Miao
Xiao-Li Xie
P2860
P2888
P304
P356
10.1007/S00109-014-1240-4
P577
2014-12-17T00:00:00Z
P5875
P6179
1053723742