Lipopolysaccharide Exposure Alleviates Asthma in Mice by Regulating Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Alleviates Asthma in Mice by Regulating Th1/Th2 and Treg/Th17 Balance.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

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P1476

Lipopolysaccharide Exposure Al ...... Th1/Th2 and Treg/Th17 Balance.

@en

P2093

Bo Liu

http://www.w3.org/2001/XMLSchema#string

Fengxia Ding

http://www.w3.org/2001/XMLSchema#string

Zhou Fu

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P2860

Endotoxin exposure and inflammation markers among agricultural workers in Colorado and Nebraska

Endotoxin Exposure during Sensitization to Blomia tropicalis Allergens Shifts TH2 Immunity Towards a TH17-Mediated Airway Neutrophilic Inflammation: Role of TLR4 and TLR2

House Dust Endotoxin Levels Are Associated with Adult Asthma in a U.S. Farming Population.

Environmental exposure to endotoxin and its relation to asthma in school-age children

Lipopolysaccharide binding protein potentiates airway reactivity in a murine model of allergic asthma.

Microbial stimulation as an aetiologic factor in atopic disease.

Concomitant exposure to ovalbumin and endotoxin augments airway inflammation but not airway hyperresponsiveness in a murine model of asthma.

Key factors in the development of asthma: atopy.

Innate Immunity and Asthma Risk in Amish and Hutterite Farm Children.

TH17 cells mediate steroid-resistant airway inflammation and airway hyperresponsiveness in mice.

P304

3220-3229

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scholarly article

P356

10.12659/MSM.905202

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P478

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P577

2018-05-16T00:00:00Z

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P698

29768397

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P921

lipopolysaccharide

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5985709

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