Vesicular glutamate release from central axons contributes to myelin damage.

about

Molecular Mechanisms of Oligodendrocyte Regeneration in White Matter-Related Diseases.Confused Connections? Targeting White Matter to Address Treatment Resistant Schizophrenia White-matter repair: Interaction between oligodendrocytes and the neurovascular unit

P2860

Q55617506-19701548-C572-43A0-B0F5-25AF1BEBD9EA Q58596335-E98833F3-2406-46AF-B8FB-2BDD136EF7C4 Q59137584-FB70818A-CD70-48FA-9DE6-34E5B35BA438

P2860

Vesicular glutamate release from central axons contributes to myelin damage.

Item

http://www.wikidata.org/entity/Q55141710

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

Vesicular glutamate release from central axons contributes to myelin damage.

@en

Vesicular glutamate release from central axons contributes to myelin damage.

@nl

type

Item

label

Vesicular glutamate release from central axons contributes to myelin damage.

@en

Vesicular glutamate release from central axons contributes to myelin damage.

@nl

prefLabel

Vesicular glutamate release from central axons contributes to myelin damage.

@en

Vesicular glutamate release from central axons contributes to myelin damage.

@nl

P1476

Vesicular glutamate release from central axons contributes to myelin damage

@en

P2093

Christian Zammit

http://www.w3.org/2001/XMLSchema#string

Daniel Bloch Hansen

http://www.w3.org/2001/XMLSchema#string

Glenn Harper

http://www.w3.org/2001/XMLSchema#string

Jasmine Vella

http://www.w3.org/2001/XMLSchema#string

Peter Bond

http://www.w3.org/2001/XMLSchema#string

P2860

Neuregulin and BDNF induce a switch to NMDA receptor-dependent myelination by oligodendrocytes

The pathobiology of vascular dementia

Developmental and regional expression in the rat brain and functional properties of four NMDA receptors

NR2B and NR2D subunits coassemble in cerebellar Golgi cells to form a distinct NMDA receptor subtype restricted to extrasynaptic sites

Mild postischemic hypothermia limits cerebral injury following transient focal ischemia in rat neocortex.

Selective suppression of excessive GluN2C expression rescues early epilepsy in a tuberous sclerosis murine model.

White matter injury in ischemic stroke.

The N-methyl-D-aspartate antagonist CNS 1102 protects cerebral gray and white matter from ischemic injury following temporary focal ischemia in rats.

In situ fluorescence imaging of myelination

Interaction of the NMDA receptor noncompetitive antagonist MK-801 with model and native membranes.

P2888

s41467-018-03427-1

P304

1032

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/S41467-018-03427-1

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Robert Fern

Mario Valentino

Sean Doyle

P577

2018-03-12T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1101436480

http://www.w3.org/2001/XMLSchema#string

P698

29531223

http://www.w3.org/2001/XMLSchema#string

P932

5847599

http://www.w3.org/2001/XMLSchema#string

Vesicular glutamate release from central axons contributes to myelin damage.

about

P2860

P2860

Vesicular glutamate release from central axons contributes to myelin damage.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P50

P577

P6179

P698

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P50

P577

P6179

P698

P932