about
The genome of the basidiomycetous yeast and human pathogen Cryptococcus neoformansAnalysis of the genome and transcriptome of Cryptococcus neoformans var. grubii reveals complex RNA expression and microevolution leading to virulence attenuation.Identification of a new family of genes involved in beta-1,2-mannosylation of glycans in Pichia pastoris and Candida albicans.Cas3p belongs to a seven-member family of capsule structure designer proteins.UGD1, encoding the Cryptococcus neoformans UDP-glucose dehydrogenase, is essential for growth at 37 degrees C and for capsule biosynthesisCapsule growth in Cryptococcus neoformans is coordinated with cell cycle progression.Cryptococcus neoformans and Cryptococcus gattii, the etiologic agents of cryptococcosis.Cryptococcus neoformans capsule structure evolution in vitro and during murine infectionCandida albicans biofilms: a developmental state associated with specific and stable gene expression patterns.Characterizing the role of RNA silencing components in Cryptococcus neoformansIdentification and functional demonstration of miRNAs in the fungus Cryptococcus neoformans.Appearance and properties of L-sorbose-utilizing mutants of Candida albicans obtained on a selective plate.Introns regulate gene expression in Cryptococcus neoformans in a Pab2p dependent pathway.Network-assisted genetic dissection of pathogenicity and drug resistance in the opportunistic human pathogenic fungus Cryptococcus neoformans.A yeast under cover: the capsule of Cryptococcus neoformans.Distinct and redundant roles of exonucleases in Cryptococcus neoformans: implications for virulence and mating.Systematic functional profiling of transcription factor networks in Cryptococcus neoformans.Combination of amphotericin B with flucytosine is active in vitro against flucytosine-resistant isolates of Cryptococcus neoformans.Murine model of dextran sulfate sodium-induced colitis reveals Candida glabrata virulence and contribution of β-mannosyltransferases.Cryptococcus neoformans capsule biosynthesis and regulation.Role of Cln1 during melanization of Cryptococcus neoformans.Monosomy of a specific chromosome determines L-sorbose utilization: a novel regulatory mechanism in Candida albicans.The Yak1 kinase is involved in the initiation and maintenance of hyphal growth in Candida albicans.Cloning and sequencing of the beta-glucosidase-encoding gene from Candida molischiana strain 35M5N.Capsular localization of the Cryptococcus neoformans polysaccharide component galactoxylomannan.Intron retention-dependent gene regulation in Cryptococcus neoformansBiofilm formation in Candida glabrata: What have we learnt from functional genomics approaches?Fungal-induced cell cycle impairment, chromosome instability and apoptosis via differential activation of NF-κB.Antigenic and biological characteristics of mutant strains of Cryptococcus neoformans lacking capsular O acetylation or xylosyl side chains.Surfactant protein D increases phagocytosis of hypocapsular Cryptococcus neoformans by murine macrophages and enhances fungal survival.O-acetylation of cryptococcal capsular glucuronoxylomannan is essential for interference with neutrophil migration.Inactivation of CaMIT1 inhibits Candida albicans phospholipomannan beta-mannosylation, reduces virulence, and alters cell wall protein beta-mannosylation.UGE1 and UGE2 regulate the UDP-glucose/UDP-galactose equilibrium in Cryptococcus neoformans.Cryptococcus neoformans senses CO2 through the carbonic anhydrase Can2 and the adenylyl cyclase Cac1The SUN41 and SUN42 genes are essential for cell separation in Candida albicans.Use of microsatellite markers and gene dosage to quantify gene copy numbers in Candida albicans.Cas1p is a membrane protein necessary for the O-acetylation of the Cryptococcus neoformans capsular polysaccharide.Isolation and characterization of capsule structure mutant strains of Cryptococcus neoformans.UV and X-ray sensitivity of Candida albicans laboratory strains and mutants having chromosomal alterations.Introns Protect Eukaryotic Genomes from Transcription-Associated Genetic Instability.
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Guilhem Janbon
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Guilhem Janbon
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Guilhem Janbon
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Guilhem Janbon
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Guilhem Janbon
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Guilhem Janbon
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0000-0002-4788-1154