Alarmin HMGB1 induces systemic and brain inflammatory exacerbation in post-stroke infection rat model.

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Alarmin HMGB1 induces systemic and brain inflammatory exacerbation in post-stroke infection rat model.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

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Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

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Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

@en

Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

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Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

@en

Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

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P1476

Alarmin HMGB1 induces systemic ...... st-stroke infection rat model.

@en

P2093

Hahnbie Lee

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Hye-Kyung Lee

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Il-Doo Kim

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Ja-Kyeong Lee

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Juli Choi

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Pyung-Lim Han

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Seung-Woo Kim

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P2860

Identification of lipopolysaccharide-binding peptide regions within HMGB1 and their effects on subclinical endotoxemia in a mouse model

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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Lipopolysaccharide from Rhodobacter sphaeroides Attenuates Microglia-Mediated Inflammation and Phagocytosis and Directs Regulatory T Cell Response

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

DAMPs, PAMPs and alarmins: all we need to know about danger

Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion.

The biology of the receptor for advanced glycation end products and its ligands.

Prolonged, 24-h delayed peripheral inflammation increases short- and long-term functional impairment and histopathological damage after focal ischemia in the rat.

Role of dihydrotestosterone in post-stroke peripheral immunosuppression after cerebral ischemia

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scholarly article

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10.1038/S41419-018-0438-8

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2018-03-19T00:00:00Z

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29555931

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5859283

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