Inhibitory effects of superoxide dismutase 3 on Propionibacterium acnes-induced skin inflammation.

about

Inhibitory effects of superoxide dismutase 3 on Propionibacterium acnes-induced skin inflammation.

Item

http://www.wikidata.org/entity/Q55259355

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@en

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@nl

type

Item

label

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@en

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@nl

prefLabel

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@en

Inhibitory effects of superoxi ...... nes-induced skin inflammation.

@nl

P1476

Inhibitory effects of superoxi ...... cnes-induced skin inflammation

@en

P2093

Christos C Zouboulis

http://www.w3.org/2001/XMLSchema#string

Shyam Kishor Sah

http://www.w3.org/2001/XMLSchema#string

Tae-Yoon Kim

http://www.w3.org/2001/XMLSchema#string

P2860

Proteomic identification of secreted proteins of Propionibacterium acnes

A systematic review and meta-analysis on Staphylococcus aureus carriage in psoriasis, acne and rosacea

Propionibacterium acnes: an underestimated pathogen in implant-associated infections

Propionibacterium acnes strain populations in the human skin microbiome associated with acne

Cellular uptake of the tat protein from human immunodeficiency virus

The role of pulsed light and heat energy (LHE) in acne clearance.

Heat-killed Propionibacterium acnes is capable of inducing inflammatory responses in skin.

Production of superoxide anions by keratinocytes initiates P. acnes-induced inflammation of the skin.

SOD3 decreases ischemic injury derived apoptosis through phosphorylation of Erk1/2, Akt, and FoxO3a.

Extracellular superoxide dismutase protects against pulmonary emphysema by attenuating oxidative fragmentation of ECM.

P304

4024

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/S41598-018-22132-Z

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Cuong Thach Nguyen

P577

2018-03-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29507345

http://www.w3.org/2001/XMLSchema#string

P932

5838256

http://www.w3.org/2001/XMLSchema#string