Inherited Immunodeficiencies With High Predisposition to Epstein–Barr Virus-Driven Lymphoproliferative Diseases.

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Inherited Immunodeficiencies With High Predisposition to Epstein–Barr Virus-Driven Lymphoproliferative Diseases.

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http://www.wikidata.org/entity/Q55261974

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2018 nî lūn-bûn

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2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

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2018年學術文章

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name

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

@en

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

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type

Item

label

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

@en

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

@nl

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Inherited Immunodeficiencies W ...... n Lymphoproliferative Diseases

@en

prefLabel

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

@en

Inherited Immunodeficiencies W ...... Lymphoproliferative Diseases.

@nl

P1476

Inherited Immunodeficiencies W ...... n Lymphoproliferative Diseases

@en

P2093

Sarah Winter

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P275

Creative Commons Attribution 4.0 International

P2860

The X-linked lymphoproliferative-disease gene product SAP regulates signals induced through the co-receptor SLAM

SAP couples Fyn to SLAM immune receptors

Lytic immune synapse function requires filamentous actin deconstruction by Coronin 1A

CTP synthase 1 deficiency in humans reveals its central role in lymphocyte proliferation

A diacylglycerol-protein kinase C-RasGRP1 pathway directs Ras activation upon antigen receptor stimulation of T cells.

Second messenger role for Mg2+ revealed by human T-cell immunodeficiency

Mg2+ regulates cytotoxic functions of NK and CD8 T cells in chronic EBV infection through NKG2D

Inactivating mutations in an SH2 domain-encoding gene in X-linked lymphoproliferative syndrome

NTB-A [correction of GNTB-A], a novel SH2D1A-associated surface molecule contributing to the inability of natural killer cells to kill Epstein-Barr virus-infected B cells in X-linked lymphoproliferative disease

X-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cells

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1103

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scholarly article

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10.3389/FIMMU.2018.01103

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English

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Sylvain Latour

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2018-06-04T00:00:00Z

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29942301

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P921

Herpesviridae infectious disease

primary immunodeficiency disease

Lymphoproliferative disorders

Epstein–Barr virus infection

Epstein–Barr virus

tumor virus infections

Añaw

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6004768

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