Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).

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Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter

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P1476

Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC)

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P2093

Daniel G Fuster

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Ganesh Pathare

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Giuseppe Albano

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P2860

Renal Ca2+ wasting, hyperabsorption, and reduced bone thickness in mice lacking TRPV5

Thiazide diuretics directly induce osteoblast differentiation and mineralized nodule formation by interacting with a sodium chloride co-transporter in bone

Vascular calcification--is aldosterone a culprit?

Regulation and function of the FGF23/klotho endocrine pathways

Dysregulation of phosphate metabolism and conditions associated with phosphate toxicity

Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism

Up-regulation of FGF23 release by aldosterone.

NFκB-sensitive Orai1 expression in the regulation of FGF23 release.

Fibroblast growth factor 23 and Klotho: physiology and pathophysiology of an endocrine network of mineral metabolism

Tumor necrosis factor-alpha: alternative role as an inhibitor of osteoclast formation in vitro.

P304

3590

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P31

scholarly article

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10.1038/S41598-018-22041-1

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Florian Lang

Manuel Anderegg

P577

2018-02-26T00:00:00Z

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P698

29483574

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P932

5826922

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