Bisecting N-Acetylglucosamine Structures Inhibit Hypoxia-Induced Epithelial-Mesenchymal Transition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine Structures Inhibit Hypoxia-Induced Epithelial-Mesenchymal Transition in Breast Cancer Cells.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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P1476

Bisecting N-Acetylglucosamine ...... sition in Breast Cancer Cells.

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P2093

Chenxing Wang

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Feng Guan

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Xiang Li

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Zengqi Tan

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P2860

Cellular response to hypoxia involves signaling via Smad proteins

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Tumor hypoxia: definitions and current clinical, biologic, and molecular aspects

HIF-1 and tumor progression: pathophysiology and therapeutics

HemI: a toolkit for illustrating heatmaps

Hypoxia--a key regulatory factor in tumour growth

Modulation of hypoxia-inducible factor 1alpha expression by the epidermal growth factor/phosphatidylinositol 3-kinase/PTEN/AKT/FRAP pathway in human prostate cancer cells: implications for tumor angiogenesis and therapeutics

Hyperoxic treatment induces mesenchymal-to-epithelial transition in a rat adenocarcinoma model.

The bisecting GlcNAc on N-glycans inhibits growth factor signaling and retards mammary tumor progression.

HER2 (neu) signaling increases the rate of hypoxia-inducible factor 1alpha (HIF-1alpha) synthesis: novel mechanism for HIF-1-mediated vascular endothelial growth factor expression.

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scholarly article

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10.3389/FPHYS.2018.00210

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2018-03-09T00:00:00Z

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29593568

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