about
Pharmacological inhibitors of TRPV4 channels reduce cytokine production, restore endothelial function and increase survival in septic miceStrain-dependent activation of NF-kappaB in the airway epithelium and its role in allergic airway inflammationRapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammationSoy biodiesel and petrodiesel emissions differ in size, chemical composition and stimulation of inflammatory responses in cells and animalsNO2 inhalation induces maturation of pulmonary CD11c+ cells that promote antigenspecific CD4+ T cell polarizationIncreased palmitate intake: higher acylcarnitine concentrations without impaired progression of β-oxidation.Tumor necrosis factor-alpha overexpression in lung disease: a single cause behind a complex phenotype.Mitochondrial ROS induced by chronic ethanol exposure promote hyper-activation of the NLRP3 inflammasome.NF-kappa B activation in airways modulates allergic inflammation but not hyperresponsivenessRelationship between synovial fluid ARGS-aggrecan fragments, cytokines, MMPs, and TIMPs following acute ACL injury: A cross-sectional study.Aligning mouse models of asthma to human endotypes of disease.Inhibition of arginase activity enhances inflammation in mice with allergic airway disease, in association with increases in protein S-nitrosylation and tyrosine nitration.The glutaredoxin/S-glutathionylation axis regulates interleukin-17A-induced proinflammatory responses in lung epithelial cells in association with S-glutathionylation of nuclear factor κB family proteins.The tick salivary protein, Salp15, inhibits the development of experimental asthmaMolecular mechanisms of nitrogen dioxide induced epithelial injury in the lung.Bone marrow-derived mesenchymal stromal cells inhibit Th2-mediated allergic airways inflammation in miceAirway epithelial indoleamine 2,3-dioxygenase inhibits CD4+ T cells during Aspergillus fumigatus antigen exposure.Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophiliaDetrimental effects of albuterol on airway responsiveness requires airway inflammation and is independent of β-receptor affinity in murine models of asthmaHistamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibilityAirway epithelial NF-κB activation promotes allergic sensitization to an innocuous inhaled antigenThe endogenous Th17 response in NO2-promoted allergic airway disease is dispensable for airway hyperresponsiveness and distinct from Th17 adoptive transfer.Reactive nitrogen species and cell signaling: implications for death or survival of lung epithelium.Inflammasome Activity in Non-Microbial Lung Inflammation.Serum amyloid A activates the NLRP3 inflammasome and promotes Th17 allergic asthma in mice.A computational model of unresolved allergic inflammation in chronic asthmaAttenuation of Th1 effector cell responses and susceptibility to experimental allergic encephalomyelitis in histamine H2 receptor knockout mice is due to dysregulation of cytokine production by antigen-presenting cells.Effects of obesity and bariatric surgery on airway hyperresponsiveness, asthma control, and inflammationHistamine H4 receptor optimizes T regulatory cell frequency and facilitates anti-inflammatory responses within the central nervous system.Reactive nitrogen species-induced cell death requires Fas-dependent activation of c-Jun N-terminal kinasePregnant serum induces neuroinflammation and seizure activity via TNFα.Susceptibility to anthrax lethal toxin is controlled by three linked quantitative trait lociObesity is associated with neutrophil dysfunction and attenuation of murine acute lung injuryProtein disulfide isomerase-endoplasmic reticulum resident protein 57 regulates allergen-induced airways inflammation, fibrosis, and hyperresponsiveness.Mouse Invariant Monoclonal Antibody NKT14: A Novel Tool to Manipulate iNKT Cell Function In Vivo.Airway epithelial regulation of allergic sensitization in asthma.Inhalation of the reactive aldehyde acrolein promotes antigen sensitization to ovalbumin and enhances neutrophilic inflammation.Lipidomic evidence that lowering the typical dietary palmitate to oleate ratio in humans decreases the leukocyte production of proinflammatory cytokines and muscle expression of redox-sensitive genes.H(1)R expression by CD11B(+) cells is not required for susceptibility to experimental allergic encephalomyelitisImmunological characteristics and management considerations in obese patients with asthma.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Matthew E Poynter
@ast
Matthew E Poynter
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Matthew E Poynter
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Matthew E Poynter
@nl
Matthew E Poynter
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type
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Matthew E Poynter
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Matthew E Poynter
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Matthew E Poynter
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Matthew E Poynter
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Matthew E Poynter
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prefLabel
Matthew E Poynter
@ast
Matthew E Poynter
@en
Matthew E Poynter
@es
Matthew E Poynter
@nl
Matthew E Poynter
@sl
P106
P31
P496
0000-0002-7578-4570